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引用本文的文献
1
Preponderance of Fis-binding sites in the R6K gamma origin and the curious effect of the penicillin resistance marker on replication of this origin in the absence of Fis.
J Bacteriol. 1996 Aug;178(16):4965-74. doi: 10.1128/jb.178.16.4965-4974.1996.
2
Binding of DnaA protein to a replication enhancer counteracts the inhibition of plasmid R6K gamma origin replication mediated by elevated levels of R6K pi protein.
J Bacteriol. 1994 Nov;176(22):6795-801. doi: 10.1128/jb.176.22.6795-6801.1994.
3
Altered (copy-up) forms of initiator protein pi suppress the point mutations inactivating the gamma origin of plasmid R6K.
J Bacteriol. 1995 Dec;177(23):6732-9. doi: 10.1128/jb.177.23.6732-6739.1995.
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Plasmid R6K DNA replication. III. Regulatory properties of the pi initiation protein.
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Plasmid R6K DNA replication. I. Complete nucleotide sequence of an autonomously replicating segment.
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Structural properties of the beta origin of replication of plasmid R6K.
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Interaction of the plasmid R6K-encoded replication initiator protein with its binding sites on DNA.
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A system to study promoter and terminator signals recognized by Escherichia coli RNA polymerase.
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Circular R-factor molecules controlling penicillinase synthesis, replicating in Escherichia coli under either relaxed or stringent control.
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