MK-801可预防甲基苯丙胺引起的纹状体多巴胺损伤，并减少细胞外多巴胺外溢。 - Suppr

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MK-801 prevents methamphetamine-induced striatal dopamine damage and reduces extracellular dopamine overflow.

作者信息

O'Dell S J, Weihmuller F B, Marshall J F

机构信息

Department of Psychobiology, University of California, Irvine 92717.

出版信息

Ann N Y Acad Sci. 1992 May 11;648:317-9. doi: 10.1111/j.1749-6632.1992.tb24567.x.

DOI:10.1111/j.1749-6632.1992.tb24567.x

PMID:1637061

Abstract

摘要

相似文献

MK-801 prevents methamphetamine-induced striatal dopamine damage and reduces extracellular dopamine overflow.

Ann N Y Acad Sci. 1992 May 11;648:317-9. doi: 10.1111/j.1749-6632.1992.tb24567.x.

Multiple methamphetamine injections induce marked increases in extracellular striatal dopamine which correlate with subsequent neurotoxicity.

Brain Res. 1991 Nov 15;564(2):256-60. doi: 10.1016/0006-8993(91)91461-9.

MK-801 protection against methamphetamine-induced striatal dopamine terminal injury is associated with attenuated dopamine overflow.

Synapse. 1992 Jun;11(2):155-63. doi: 10.1002/syn.890110209.

MK-801, a non-competitive antagonist of NMDA receptor, prevents methamphetamine-induced decrease of striatal dopamine uptake sites in the rat striatum.

Neurosci Lett. 1992 Feb 17;136(1):39-42. doi: 10.1016/0304-3940(92)90642-k.

Presynaptic L-type Ca(2)+ channels on excessive dopamine release from rat caudate putamen.

Physiol Behav. 2000 Mar;68(5):641-9. doi: 10.1016/s0031-9384(99)00227-9.

MK-801 fails to modify the effect of methamphetamine on dopamine release in the rat striatum.

Neuroreport. 1991 May;2(5):236-8. doi: 10.1097/00001756-199105000-00005.

Dopaminergic transplants suppress L-DOPA-induced Fos expression in the dopamine-depleted striatum in a rat model of Parkinson's disease.

Brain Res. 1996 Jul 15;727(1-2):205-11. doi: 10.1016/0006-8993(96)00381-2.

Pharmacologic modulation of MPTP toxicity: MK 801 in prevention of dopaminergic cell death in monkeys and mice.

Ann N Y Acad Sci. 1992 May 11;648:268-71. doi: 10.1111/j.1749-6632.1992.tb24553.x.

Long-term methamphetamine-induced decreases of [(11)C]WIN 35,428 binding in striatum are reduced by GDNF: PET studies in the vervet monkey.

Synapse. 2000 Mar 15;35(4):243-9. doi: 10.1002/(SICI)1098-2396(20000315)35:4<243::AID-SYN1>3.0.CO;2-N.

Dopamine-glutamate interactions in methamphetamine-induced neurotoxicity.

J Neural Transm Gen Sect. 1993;91(2-3):241-54. doi: 10.1007/BF01245234.

引用本文的文献

The role of the neuropeptide somatostatin on methamphetamine and glutamate-induced neurotoxicity in the striatum of mice.

Brain Res. 2013 May 13;1510:38-47. doi: 10.1016/j.brainres.2013.03.010. Epub 2013 Mar 19.

Glial reactivity in resistance to methamphetamine-induced neurotoxicity.

J Neurochem. 2013 May;125(4):566-74. doi: 10.1111/jnc.12201. Epub 2013 Mar 17.

The neurokinin-1 receptor modulates the methamphetamine-induced striatal apoptosis and nitric oxide formation in mice.

J Neurochem. 2009 Nov;111(3):656-68. doi: 10.1111/j.1471-4159.2009.06330.x. Epub 2009 Aug 13.

Induction of striatal pre- and postsynaptic damage by methamphetamine requires the dopamine receptors.

Synapse. 2005 Nov;58(2):110-21. doi: 10.1002/syn.20185.

High-dose methamphetamine acutely activates the striatonigral pathway to increase striatal glutamate and mediate long-term dopamine toxicity.

J Neurosci. 2004 Dec 15;24(50):11449-56. doi: 10.1523/JNEUROSCI.3597-04.2004.

Antagonists of the neurokinin-1 or dopamine D1 receptors confer protection from methamphetamine on dopamine terminals of the mouse striatum.

Ann N Y Acad Sci. 2004 Oct;1025:171-80. doi: 10.1196/annals.1316.022.

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MK-801 prevents methamphetamine-induced striatal dopamine damage and reduces extracellular dopamine overflow.

作者信息

O'Dell S J, Weihmuller F B, Marshall J F

机构信息

Department of Psychobiology, University of California, Irvine 92717.

出版信息

Ann N Y Acad Sci. 1992 May 11;648:317-9. doi: 10.1111/j.1749-6632.1992.tb24567.x.

DOI:10.1111/j.1749-6632.1992.tb24567.x

PMID:1637061

Abstract

摘要

相似文献

MK-801 prevents methamphetamine-induced striatal dopamine damage and reduces extracellular dopamine overflow.

Ann N Y Acad Sci. 1992 May 11;648:317-9. doi: 10.1111/j.1749-6632.1992.tb24567.x.

Multiple methamphetamine injections induce marked increases in extracellular striatal dopamine which correlate with subsequent neurotoxicity.

Brain Res. 1991 Nov 15;564(2):256-60. doi: 10.1016/0006-8993(91)91461-9.

MK-801 protection against methamphetamine-induced striatal dopamine terminal injury is associated with attenuated dopamine overflow.

Synapse. 1992 Jun;11(2):155-63. doi: 10.1002/syn.890110209.

MK-801, a non-competitive antagonist of NMDA receptor, prevents methamphetamine-induced decrease of striatal dopamine uptake sites in the rat striatum.

Neurosci Lett. 1992 Feb 17;136(1):39-42. doi: 10.1016/0304-3940(92)90642-k.

Presynaptic L-type Ca(2)+ channels on excessive dopamine release from rat caudate putamen.

Physiol Behav. 2000 Mar;68(5):641-9. doi: 10.1016/s0031-9384(99)00227-9.

MK-801 fails to modify the effect of methamphetamine on dopamine release in the rat striatum.

Neuroreport. 1991 May;2(5):236-8. doi: 10.1097/00001756-199105000-00005.

Dopaminergic transplants suppress L-DOPA-induced Fos expression in the dopamine-depleted striatum in a rat model of Parkinson's disease.

Brain Res. 1996 Jul 15;727(1-2):205-11. doi: 10.1016/0006-8993(96)00381-2.

Pharmacologic modulation of MPTP toxicity: MK 801 in prevention of dopaminergic cell death in monkeys and mice.

Ann N Y Acad Sci. 1992 May 11;648:268-71. doi: 10.1111/j.1749-6632.1992.tb24553.x.

Long-term methamphetamine-induced decreases of [(11)C]WIN 35,428 binding in striatum are reduced by GDNF: PET studies in the vervet monkey.

Synapse. 2000 Mar 15;35(4):243-9. doi: 10.1002/(SICI)1098-2396(20000315)35:4<243::AID-SYN1>3.0.CO;2-N.

Dopamine-glutamate interactions in methamphetamine-induced neurotoxicity.

J Neural Transm Gen Sect. 1993;91(2-3):241-54. doi: 10.1007/BF01245234.

引用本文的文献

The role of the neuropeptide somatostatin on methamphetamine and glutamate-induced neurotoxicity in the striatum of mice.

Brain Res. 2013 May 13;1510:38-47. doi: 10.1016/j.brainres.2013.03.010. Epub 2013 Mar 19.

Glial reactivity in resistance to methamphetamine-induced neurotoxicity.

J Neurochem. 2013 May;125(4):566-74. doi: 10.1111/jnc.12201. Epub 2013 Mar 17.

The neurokinin-1 receptor modulates the methamphetamine-induced striatal apoptosis and nitric oxide formation in mice.

J Neurochem. 2009 Nov;111(3):656-68. doi: 10.1111/j.1471-4159.2009.06330.x. Epub 2009 Aug 13.

Induction of striatal pre- and postsynaptic damage by methamphetamine requires the dopamine receptors.

Synapse. 2005 Nov;58(2):110-21. doi: 10.1002/syn.20185.

High-dose methamphetamine acutely activates the striatonigral pathway to increase striatal glutamate and mediate long-term dopamine toxicity.

J Neurosci. 2004 Dec 15;24(50):11449-56. doi: 10.1523/JNEUROSCI.3597-04.2004.

Antagonists of the neurokinin-1 or dopamine D1 receptors confer protection from methamphetamine on dopamine terminals of the mouse striatum.

Ann N Y Acad Sci. 2004 Oct;1025:171-80. doi: 10.1196/annals.1316.022.