肝腺瘤样增生在动脉门静脉造影CT上表现为高密度：病例报告

Hepatic adenomatous hyperplasia with hyperattenuation on CT during arterial portography: a case report.

作者信息

Sato Shuzo, Rai Tsuyoshi, Ohira Hiromasa, Abe Kazumichi, Saito Hironobu, Takahashi Atsushi, Sakamoto Natsumi, Hoshi Namiko, Suzuki Eiji, Sato Yukio

机构信息

Department of Internal Medicine II, Fukushima Medical University School of Medicine, Fukushima, 960-1295, Japan.

出版信息

Fukushima J Med Sci. 2005 Dec;51(2):87-94. doi: 10.5387/fms.51.87.

DOI:10.5387/fms.51.87

PMID:16555629

Abstract

We report a 72-year-old man who was admitted to our department with multiple nodules of hepatocellular carcinoma (HCC) in a cirrhotic liver because of HCV infection. Unlike most of the nodules, one in segment 2 (S2) showed hypoattenuation on computed tomography (CT) during hepatic arteriography (CTA), and hyperattenuation on CT during arterial portography (CTAP). Fine needle aspiration biopsy of the nodule established the diagnosis of hepatic adenomatous hyperplasia. Why the S2 nodule showed hyperattenuation on CTAP is not clear. Two possibilities are considered: i) greater portal blood flow into the nodule than into the surrounding cirrhotic parenchyma, ii) existence of a period during the course of hepatocarcinogenesis when the portal blood flow into the nodule is higher in density on CTAP.

摘要

我们报告一名72岁男性，因丙型肝炎病毒（HCV）感染导致肝硬化肝脏出现多个肝细胞癌（HCC）结节而入住我科。与大多数结节不同，肝动脉造影（CTA）期间，2段（S2）的一个结节在计算机断层扫描（CT）上表现为低密度，而在动脉门静脉造影（CTAP）期间，CT上表现为高密度。对该结节进行细针穿刺活检确诊为肝腺瘤样增生。S2结节在CTAP上为何表现为高密度尚不清楚。考虑有两种可能性：i）进入结节的门静脉血流多于进入周围肝硬化实质的血流；ii）在肝癌发生过程中，存在门静脉血流在CTAP上密度较高的时期。