一名血液透析患者的代谢性碱中毒——质子泵抑制剂治疗成功
Metabolic alkalosis in a hemodialysis patient--successful treatment with a proton pump inhibitor.
作者信息
Kirsch B M, Sunder-Plassmann G, Schwarz C
机构信息
Division of Nephrology and Dialysis, Department of Medicine Ill, Medical University of Vienna, Austria.
出版信息
Clin Nephrol. 2006 Nov;66(5):391-4. doi: 10.5414/cnp66391.
Hemodialysis patients develop metabolic acidosis due to their impaired excretion of daily produced protons (H+). The following report will show a rare case of severe metabolic alkalosis (predialysis pH 7.52, base excess (BE) +17) in a hemodialysis woman caused by self-provoked upper gastrointestinal H+ losses based on an eating disorder. Treatment with a proton pump inhibitor resulted in the normalization of acid/base homeostasis (predialysis pH 7.40, BE +1.6).
血液透析患者由于每日产生的质子（H+）排泄受损而发生代谢性酸中毒。以下报告将展示一例罕见的严重代谢性碱中毒病例（透析前pH值7.52，碱剩余（BE）+17），该病例发生在一名血液透析女性患者身上，病因是基于饮食失调导致的自身诱发的上消化道H+丢失。使用质子泵抑制剂治疗后，酸碱平衡恢复正常（透析前pH值7.40，BE +1.6）。
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