CD44缺陷是儿童伯基特淋巴瘤和白血病中一致的发现。 - Suppr

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超能文献

CD44 deficiency is a consistent finding in childhood Burkitt's lymphoma and leukemia.

作者信息

Attarbaschi A, Mann G, Schumich A, König M, Pickl W F, Haas O A, Gadner H, Dworzak M N

出版信息

Leukemia. 2007 May;21(5):1110-3. doi: 10.1038/sj.leu.2404608. Epub 2007 Mar 1.

DOI:10.1038/sj.leu.2404608

PMID:17330102

Abstract

摘要

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CD44 deficiency is a consistent finding in childhood Burkitt's lymphoma and leukemia.CD44缺陷是儿童伯基特淋巴瘤和白血病中一致的发现。

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Homogeneous immunophenotype and paucity of secondary genomic aberrations are distinctive features of endemic but not of sporadic Burkitt's lymphoma and diffuse large B-cell lymphoma with MYC rearrangement.均匀的免疫表型和继发性基因组畸变的缺乏是地方性伯基特淋巴瘤的显著特征，但不是伴有MYC重排的散发性伯基特淋巴瘤和弥漫性大B细胞淋巴瘤的特征。

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A biologic definition of Burkitt's lymphoma from transcriptional and genomic profiling.基于转录组和基因组分析的伯基特淋巴瘤生物学定义

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Activation of the c-myc p1 promoter in Burkitt's lymphoma by the hs3 immunoglobulin heavy-chain gene enhancer.hs3免疫球蛋白重链基因增强子对伯基特淋巴瘤中c-myc p1启动子的激活作用。

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Burkitt's lymphomas express VH genes with a moderate number of antigen-selected somatic mutations.伯基特淋巴瘤表达具有中等数量抗原选择体细胞突变的VH基因。

Am J Pathol. 1995 Nov;147(5):1398-407.

The intron enhancer of the immunoglobulin kappa gene activates c-myc but does not induce the Burkitt-specific promoter shift.免疫球蛋白κ基因的内含子增强子激活c-myc，但不诱导伯基特淋巴瘤特异性的启动子移位。

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Level of MYC overexpression in pediatric Burkitt's lymphoma is strongly dependent on genomic breakpoint location within the MYC locus.小儿伯基特淋巴瘤中MYC过表达水平强烈依赖于MYC基因座内的基因组断点位置。

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Identification of two distinct MYC breakpoint clusters and their association with various IGH breakpoint regions in the t(8;14) translocations in sporadic Burkitt-lymphoma.散发性伯基特淋巴瘤中t(8;14)易位中两个不同的MYC断点簇的鉴定及其与各种IGH断点区域的关联

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Study of the HLA system in Burkitt's lymphoma.伯基特淋巴瘤中HLA系统的研究。

Hum Immunol. 1980 Oct;1(3):207-10. doi: 10.1016/0198-8859(80)90015-4.

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