Suppr超能文献

相似文献

1
Induction of high STAT1 expression in transgenic mice with LQTS and heart failure.
Biochem Biophys Res Commun. 2007 Jun 29;358(2):449-54. doi: 10.1016/j.bbrc.2007.04.119. Epub 2007 Apr 26.
3
Optical mapping of ventricular arrhythmias in LQTS mice with SCN5A mutation N1325S.
Biochem Biophys Res Commun. 2007 Jan 26;352(4):879-83. doi: 10.1016/j.bbrc.2006.11.106. Epub 2006 Dec 1.
4
Mutation analysis in congenital Long QT Syndrome--a case with missense mutations in KCNQ1 and SCN5A.
Genet Test. 2003 Spring;7(1):57-61. doi: 10.1089/109065703321560958.
5
Functional suppression of Kcnq1 leads to early sodium channel remodelling and cardiac conduction system dysmorphogenesis.
Cardiovasc Res. 2013 Jun 1;98(3):504-14. doi: 10.1093/cvr/cvt076. Epub 2013 Mar 29.
6
Cardiac-specific overexpression of SCN5A gene leads to shorter P wave duration and PR interval in transgenic mice.
Biochem Biophys Res Commun. 2007 Apr 6;355(2):444-50. doi: 10.1016/j.bbrc.2007.01.170. Epub 2007 Feb 7.
7
Regulation of myocardial interleukin-6 expression by p53 and STAT1.
J Interferon Cytokine Res. 2013 Sep;33(9):542-8. doi: 10.1089/jir.2012.0165. Epub 2013 May 15.
10
A revised view of cardiac sodium channel "blockade" in the long-QT syndrome.
J Clin Invest. 2000 Apr;105(8):1133-40. doi: 10.1172/JCI9212.

引用本文的文献

2
Biophysical mechanisms of myocardium sodium channelopathies.
Pflugers Arch. 2024 May;476(5):735-753. doi: 10.1007/s00424-024-02930-3. Epub 2024 Mar 1.
4
CCND1 Overexpression in Idiopathic Dilated Cardiomyopathy: A Promising Biomarker?
Genes (Basel). 2023 Jun 10;14(6):1243. doi: 10.3390/genes14061243.
6
Identification of potentially relevant genes for myocardial infarction using RNA sequencing data analysis.
Exp Ther Med. 2018 Feb;15(2):1456-1464. doi: 10.3892/etm.2017.5580. Epub 2017 Nov 28.
7
Nemo-Like Kinase (NLK) Is a Pathological Signaling Effector in the Mouse Heart.
PLoS One. 2016 Oct 20;11(10):e0164897. doi: 10.1371/journal.pone.0164897. eCollection 2016.
8
Coherent functional modules improve transcription factor target identification, cooperativity prediction, and disease association.
PLoS Genet. 2014 Feb 6;10(2):e1004122. doi: 10.1371/journal.pgen.1004122. eCollection 2014 Feb.

本文引用的文献

2
Cardiac-specific overexpression of SCN5A gene leads to shorter P wave duration and PR interval in transgenic mice.
Biochem Biophys Res Commun. 2007 Apr 6;355(2):444-50. doi: 10.1016/j.bbrc.2007.01.170. Epub 2007 Feb 7.
3
Optical mapping of ventricular arrhythmias in LQTS mice with SCN5A mutation N1325S.
Biochem Biophys Res Commun. 2007 Jan 26;352(4):879-83. doi: 10.1016/j.bbrc.2006.11.106. Epub 2006 Dec 1.
4
Characterization of the cardiac sodium channel SCN5A mutation, N1325S, in single murine ventricular myocytes.
Biochem Biophys Res Commun. 2007 Jan 12;352(2):378-83. doi: 10.1016/j.bbrc.2006.11.019. Epub 2006 Nov 14.
5
Sodium channel mutations and susceptibility to heart failure and atrial fibrillation.
JAMA. 2005 Jan 26;293(4):447-54. doi: 10.1001/jama.293.4.447.
6
Expression profiling of cardiovascular disease.
Hum Genomics. 2004 Aug;1(5):355-70. doi: 10.1186/1479-7364-1-5-355.
7
Post-transcriptional alterations in the expression of cardiac Na+ channel subunits in chronic heart failure.
J Mol Cell Cardiol. 2004 Jul;37(1):91-100. doi: 10.1016/j.yjmcc.2004.04.003.
8
STAT-1: a novel regulator of apoptosis.
Int J Exp Pathol. 2003 Dec;84(6):239-44. doi: 10.1111/j.0959-9673.2003.00363.x.
9
Mechanisms by which SCN5A mutation N1325S causes cardiac arrhythmias and sudden death in vivo.
Cardiovasc Res. 2004 Feb 1;61(2):256-67. doi: 10.1016/j.cardiores.2003.11.007.
10
Identification of new genes differentially expressed in coronary artery disease by expression profiling.
Physiol Genomics. 2003 Sep 29;15(1):65-74. doi: 10.1152/physiolgenomics.00181.2002.

文献AI研究员

20分钟写一篇综述,助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型,支持多种主流文档格式。

立即体验