Nordland O S
Can J Comp Med Vet Sci. 1960 Mar;24(3):88-95.
Experimental hyperglycemia and infection with L. monocytogenes, and the effects of somatotsopic hormone and cortisone, were studied. Hamsters, obese mice, and normal mice were used. The results supported the hypothesis that an upset carbohydrate metabolism initiated by stress is the trigger mechanism that makes conditions favourable for proliferation of L. monocytogenes and probably of other glucose fermenting pathogenic agents.
研究了实验性高血糖和单核细胞增生李斯特菌感染,以及生长激素和可的松的作用。使用了仓鼠、肥胖小鼠和正常小鼠。结果支持了以下假设:应激引发的碳水化合物代谢紊乱是一种触发机制,使得单核细胞增生李斯特菌以及可能其他葡萄糖发酵致病因子的增殖条件变得有利。