Thomas R Z, Ruben J L, ten Bosch J J, Fidler V, Huysmans M C D N J M
Department of Dentistry and Dental Hygiene, University Medical Center Groningen, University of Groningen, Groningen, The Netherlands.
Caries Res. 2007;41(5):399-405. doi: 10.1159/000104799.
There is no consensus about the definition and progression of outer and wall lesions in secondary caries. In this study we investigated whether lesion progression is influenced by an adjacent composite restoration and whether wall lesions develop at the composite-tooth interface. In order to study the appearance and progression of approximal primary caries lesions and lesions next to composite restorations, 16 samples were placed in a full denture of each of 8 subjects. Each denture housed 4 restored and 4 unrestored enamel samples and similarly 8 dentin samples. All samples were distributed over 2 sample holders, in each of which 4 approximal spaces were simulated. Every 4 weeks the sample holders were microradiographed using transversal wavelength independent microradiography and lesion depth was measured. At the end of the study, after 20 weeks, the lesion depth of the outer lesions was 0-350 microm for enamel and 0-750 microm for dentin. The estimated difference in progression between secondary and primary lesions (1.1 microm/4 weeks, 95% CI: -9.2 to 11.4 microm) was not statistically significant (p = 0.83). Secondary outer lesions appeared and progressed as primary caries lesions. No clear wall lesions were found next to composite, but they were observed next to acrylic resin.
关于继发龋中外层和洞壁病变的定义及进展尚无共识。在本研究中,我们调查了病变进展是否受相邻复合树脂修复体的影响,以及洞壁病变是否在复合树脂与牙齿的界面处形成。为了研究邻面原发性龋损及复合树脂修复体旁病变的外观和进展情况,在8名受试者的每副全口义齿中放置了16个样本。每副义齿包含4个修复后的釉质样本和4个未修复的釉质样本,以及同样数量的8个牙本质样本。所有样本分布在2个样本架上,每个样本架模拟4个邻面间隙。每4周使用横向波长无关显微放射摄影对样本架进行显微放射照相,并测量病变深度。在研究结束时,即20周后,外层病变的釉质病变深度为0 - 350微米,牙本质病变深度为0 - 750微米。继发龋损与原发性龋损进展的估计差异(1.1微米/4周,95%可信区间:-9.2至11.4微米)无统计学意义(p = 0.83)。继发外层病变的出现和进展与原发性龋损相同。在复合树脂旁未发现明显的洞壁病变,但在丙烯酸树脂旁观察到了洞壁病变。