Jonsson L, Thuomas K A, Stenquist M, Engström M, Stålberg E, Bergström K, Lyttkens L, Svedberg A
Department of Otolaryngology, Uppsala University, Akademiska Sjukhuset, Sweden.
ORL J Otorhinolaryngol Relat Spec. 1991;53(6):362-5. doi: 10.1159/000276248.
A transient pontine lesion was demonstrated in a young adult male who had a complete acute peripheral facial nerve palsy due probably to multiple sclerosis. In the acute stage of the palsy. T2-weighted magnetic resonance imaging (MRI) revealed a high signal intensity in the ipsilateral pons in the region of the nucleus and pontine part of the facial nerve. The patient recovered completely 5 weeks after the onset of the palsy, and at this stage, the lesion in the brainstem was no longer demonstrable on MRI. The onset and course of the disease resembled the idiopathic form of facial palsy (Bell's palsy): the present findings along with recent MRI and topodiagnostic studies may indicate that in some cases of Bell's palsy the primary lesion is located centrally. By improved MRI techniques and intravenous contrast agents it may be possible to visualize and follow the disease process and ascertain more of the pathogenesis of Bell's palsy.
一名年轻成年男性被证实存在短暂性脑桥病变，该患者因多发性硬化症可能导致了完全性急性周围性面神经麻痹。在麻痹的急性期，T2加权磁共振成像（MRI）显示面神经核及脑桥部分同侧脑桥区域有高信号强度。患者在麻痹发作5周后完全康复，此时，脑干病变在MRI上已不再显示。该疾病的起病和病程类似于特发性面神经麻痹（贝尔麻痹）：目前的发现以及最近的MRI和局部诊断研究可能表明，在某些贝尔麻痹病例中，原发性病变位于中枢。通过改进的MRI技术和静脉造影剂，有可能观察并跟踪疾病进程，进一步明确贝尔麻痹的发病机制。
