Pedra Carlos A C, Pontes Sérgio C, Pedra Simone R F, Salerno Lucia, Sousa J Breno, Miaira Marly A, Guerra Ana Luisa, Santana M Virginia T, Silva M Aparecida, Fontes Valmir F
Catheterization Laboratory for Congenital Heart Disease, Instituto Dante Pazzanese de Cardiologia, São Paulo, SP, Brazil.
J Invasive Cardiol. 2007 Nov;19(11):491-5.
Although uncommon, significant postoperative residual leaks may occur after repair of any type of ventricular septal defect (VSD). Post-traumatic VSDs are even rarer, but can be occasionally seen after penetrating or blunt chest trauma. When these defects are associated with significant left-to-right shunting (Qp/Qs > 1.5) with persistent left ventricular volume overload, intervention is generally recommended. Surgical treatment requires cardiopulmonary bypass with its attendant morbidity, increased hospital stay and possible long-term neurological impairment. With the evolving experience of transcatheter closure of postinfarction and native perimembranous and muscular VSDs, this less invasive method became an attractive alternative to manage these postoperative and post-traumatic defects. In this paper, we report on 3 patients with such residual leaks after repair of a perimembranous defect, which was closed using a perimembranous Amplatzer VSD occluded after a failed attempt using a NitOcclud coil. One patient had a mid-muscular post-traumatic defect after a penetrating chest wound, and another patient had a residual leak after a patch repair of a large post-traumatic muscular defect with outlet extension after a blunt chest trauma. Both defects were closed using muscular Amplatzer VSD occluders. All procedures were uncomplicated, and there were no technical difficulties with device implantation. All 3 patients' defects were completely closed at follow up. Percutaneous closure of traumatic and residual postoperative VSDs appears to be safe and effective. A larger number of patients and longer follow-up period are needed before the widespread use of this technique can be recommended.
尽管不常见,但任何类型的室间隔缺损(VSD)修复术后都可能发生显著的术后残余分流。创伤后室间隔缺损更为罕见,但在穿透性或钝性胸部创伤后偶尔可见。当这些缺损伴有显著的左向右分流(Qp/Qs > 1.5)且左心室容量持续超负荷时,一般建议进行干预。手术治疗需要体外循环,随之而来的是发病率增加、住院时间延长以及可能的长期神经功能损害。随着经导管封堵心肌梗死后、先天性膜周部和肌部室间隔缺损经验的不断积累,这种侵入性较小的方法成为处理这些术后和创伤后缺损的一种有吸引力的替代方案。在本文中,我们报告了3例膜周部缺损修复术后出现此类残余分流的患者,其中1例在使用NitOcclud线圈封堵失败后,采用膜周部Amplatzer室间隔缺损封堵器成功封堵;1例在穿透性胸部创伤后出现肌部中间创伤后缺损,另1例在钝性胸部创伤后出现大的创伤后肌部缺损伴流出道延伸,经补片修复后仍有残余分流,这2例缺损均采用肌部Amplatzer室间隔缺损封堵器成功封堵。所有手术均顺利,器械植入无技术困难。3例患者的缺损在随访时均完全闭合。经皮封堵创伤性和术后残余室间隔缺损似乎是安全有效的。在推荐广泛应用该技术之前,还需要更多的患者和更长的随访时间。