相似文献
1
Inhibition of pyruvate oxidation in the malarial parasite Plasmodium gallinaceum by quinine treatment of the host.
J Infect Dis. 1949 Sep-Oct;85(2):195-204. doi: 10.1093/infdis/85.2.195.
2
Effect of quinine treatment of the host upon the carbohydrate metabolism of the malarial parasite Plasmodium gallinaceum.
J Infect Dis. 1948 Nov-Dec;83(3):262-70. doi: 10.1093/infdis/83.3.262.
3
Studies on parasite-host interplay between Plasmodium gallinaceum and the chicken as influenced by hydroxynaphthoquinones.
J Infect Dis. 1948 Mar-Apr;82(2):138-62. doi: 10.1093/infdis/82.2.138.
4
The biochemistry of the malaria parasite; the in vitro effects of x-rays upon Plasmodium gallinaceum.
J Infect Dis. 1946 May-Jun;78:190-7. doi: 10.1093/infdis/78.3.190.
5
Acquired resistance (twofold) to quinine in Plasmodium gallinaceum.
Nature. 1947 Nov 1;160(4070):606. doi: 10.1038/160606a0.
6
Role of quinine hydrochloride treatment in modifying the pathological processes in Plasmodium gallinaceum infections in chicks.
Exp Parasitol. 1962 Jun;12:164-7. doi: 10.1016/0014-4894(62)90053-x.
7
Absence of potentiation between quinine and pyrimethamine in infections of Plasmodium gallinaceum in chicks.
Parasitology. 1956 May;46(1-2):220-3.
8
Twofold quinine resistance of Plasmodium gallinaceum, induced by regular administration of the drug.
Doc Neerl Indones Morbis Trop. 1949 Mar;1(1):55-66.
9
Acquired resistance against quinine in plasmodium gallinaceum.
Acta Brevia Neerl Physiol Pharmacol Microbiol E A. 1948;16(1-4):34.
10
De novo assembly and transcriptome analysis of Plasmodium gallinaceum identifies the Rh5 interacting protein (ripr), and reveals a lack of EBL and RH gene family diversification.
Malar J. 2015 Aug 5;14:296. doi: 10.1186/s12936-015-0814-0.
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