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Does inhibition of glycogen synthase kinase protect in mice?

作者信息

Murphy Elizabeth, Steenbergen Charles

机构信息

Vascular Medicine Branch, National Heart, Lung, and Blood Institute, NIH, Bethesda, MD 20892, USA.

出版信息

Circ Res. 2008 Aug 1;103(3):226-8. doi: 10.1161/CIRCRESAHA.108.181602.

Abstract
摘要

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本文引用的文献

1
Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse.
Circ Res. 2008 Aug 1;103(3):307-14. doi: 10.1161/CIRCRESAHA.107.169953. Epub 2008 Jun 26.
2
Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion.
Circulation. 2008 May 27;117(21):2761-8. doi: 10.1161/CIRCULATIONAHA.107.755066. Epub 2008 May 19.
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Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death.
Nat Cell Biol. 2007 May;9(5):550-5. doi: 10.1038/ncb1575. Epub 2007 Apr 8.
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Preconditioning: the mitochondrial connection.
Annu Rev Physiol. 2007;69:51-67. doi: 10.1146/annurev.physiol.69.031905.163645.
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Opioid-induced cardioprotection occurs via glycogen synthase kinase beta inhibition during reperfusion in intact rat hearts.
Circ Res. 2004 Apr 16;94(7):960-6. doi: 10.1161/01.RES.0000122392.33172.09. Epub 2004 Feb 19.

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