Arangio George A, Salathe Eric P
Penn State, Milton S. Hershey Medical College, Department of Surgery, Division of Orthopedic Surgery, Lehigh Valley Hospital, Allentown, PA 18103, USA.
Clin Biomech (Bristol). 2009 May;24(4):385-90. doi: 10.1016/j.clinbiomech.2009.01.009. Epub 2009 Mar 9.
Biomechanical models have been used to study stress in the metatarsals, subtalar motion, lateral column lengthening and subtalar arthroereisis. Posterior tibial tendon dysfunction has been associated with increased loads in the arch of the acquired flat foot. We examine whether a 10 millimeter (mm) medial displacement calcaneal osteotomy and flexor digitorum longus transfer to the navicular reduces these increased loads in the flat foot.
The response of a normal foot, a foot with posterior tibial tendon dysfunction, and a flat foot to an applied load of 683Newton was analyzed using a multi-segment biomechanical model. The distribution of load on the metatarsals, the moment about each joint, the force on each of the plantar ligaments and the muscle forces were computed.
Posterior tibial tendon dysfunction results in increased load on the medial arch, which may cause the foot to flatten. A 10mm medial displacement calcaneal osteotomy substantially decreases the load on the first metatarsal and the moment at the talo-navicular joint and increases the load on the fifth metatarsal and the calcaneal-cuboid joint. Adding the flexor digitorum longus transfer to the medial displacement calcaneal osteotomy has only a small effect on the flattened foot.
Our biomechanical analysis illustrates that when the foot becomes flat, the force on the talo-navicular joint increases substantially from its value for the normal foot, and that medial displacement calcaneal osteotomy can reduce this increased force back toward the value occurring in the normal foot. This study provides a biomechanical rationale for medial displacement calcaneal osteotomy treatments for posterior tibial tendon dysfunction.
生物力学模型已被用于研究跖骨应力、距下关节运动、外侧柱延长和距下关节制动术。胫后肌腱功能障碍与后天性平足足弓负荷增加有关。我们研究了跟骨内侧移位截骨术(10毫米)联合趾长屈肌转移至舟骨是否能减轻平足中这些增加的负荷。
使用多节段生物力学模型分析正常足、胫后肌腱功能障碍足和平足在683牛顿外加负荷下的反应。计算跖骨上的负荷分布、每个关节的力矩、每条足底韧带的受力以及肌肉力量。
胫后肌腱功能障碍导致内侧足弓负荷增加,这可能导致足扁平。跟骨内侧移位截骨术(10毫米)可显著降低第一跖骨上的负荷以及距舟关节处的力矩,并增加第五跖骨和跟骰关节上的负荷。在跟骨内侧移位截骨术基础上增加趾长屈肌转移对扁平足的影响较小。
我们的生物力学分析表明,当足变得扁平,距舟关节上的力相较于正常足大幅增加,而跟骨内侧移位截骨术可将这种增加的力降低至接近正常足的水平。本研究为胫后肌腱功能障碍的跟骨内侧移位截骨术治疗提供了生物力学依据。