Sampaio Sergio M, Shin Susanna H, Panneton Jean M, Andrews James C, Bower Thomas C, Cherry Kenneth J, Duncan Audra A, Kalra Manju, Gloviczki Peter
Eastern Virginia Medical School, and Division of Vascular Surgery, Sentara Heart Hospital, Norfolk, Virginia, USA.
Vasc Endovascular Surg. 2009 Aug-Sep;43(4):352-9. doi: 10.1177/1538574409333581. Epub 2009 Apr 7.
Endoleaks are critical complications of endovascular abdominal aortic aneurysm repair (EVAR). This study sought to determine the frequency and nature of intraoperative endoleaks and their impact on postoperative endoleak-related events.
A retrospective chart review was performed of all patients who underwent EVAR at our institution. The impact of intraoperative endoleaks on postoperative endoleak rates and endoleak-related reintervention rates were assessed.
From December 18, 1996, to May 21, 2003, 241 patients underwent EVAR. An endoleak was observed during 126 (52.3%) procedures. Type I endoleaks were observed in 63 (26.1%) cases: 35 proximal and 31 distal endoleaks (3 cases at both attachments). Angioplasty, additional cuff placement, or stenting corrected 59 (89.4%) of these endoleaks. A total of 71 type II intraoperative endoleaks (29.5%) and 8 type IV endoleaks (3.3%) were observed without any attempted corrective maneuvers. Ten type III endoleaks (4.2%) occurred but all resolved with angioplasty or additional cuff placement. In all, 86 (35.7%) endoleaks persisted on completion angiogram. Patients with a type I or type II intraoperative endoleak were more likely to have an endoleak at 1.5 years (31.4% vs. 21.6%, P=.018). Reinterventions were required more often after an intraoperative type I endoleak (10% vs. 4%, P=.003). Patients with intraoperative endoleaks demonstrated a trend toward less postoperative aneurysm diameter reduction at 2 years (43.8% vs. 74.5%, P=.104).
The presence of a type I or a type II endoleak during EVAR significantly increases the likelihood of a postoperative endoleak and should prompt a high degree of suspicion during follow-up.
内漏是血管腔内腹主动脉瘤修复术(EVAR)的关键并发症。本研究旨在确定术中内漏的发生率、性质及其对术后内漏相关事件的影响。
对在本机构接受EVAR的所有患者进行回顾性病历审查。评估术中内漏对术后内漏发生率和内漏相关再次干预率的影响。
1996年12月18日至2003年5月21日,241例患者接受了EVAR。126例(52.3%)手术过程中观察到内漏。63例(26.1%)观察到I型内漏:35例近端内漏和31例远端内漏(3例在两个附着点均有)。血管成形术、额外放置袖带或支架纠正了其中59例(89.4%)内漏。共观察到71例II型术中内漏(29.5%)和8例IV型内漏(3.3%),未尝试任何纠正措施。发生10例III型内漏(4.2%),但均通过血管成形术或额外放置袖带得以解决。总共86例(35.7%)内漏在完成血管造影时持续存在。术中发生I型或II型内漏的患者在1.5年时更有可能出现内漏(31.4%对21.6%,P = 0.018)。术中发生I型内漏后更常需要再次干预(10%对4%,P = 0.003)。术中发生内漏的患者在2年时术后动脉瘤直径缩小的趋势较小(43.8%对74.5%,P = 0.104)。
EVAR过程中存在I型或II型内漏会显著增加术后内漏的可能性,在随访期间应引起高度怀疑。