Calabrò Maria Pia, Cerrito Marco, Luzza Francesco, Oreto Giuseppe
Department of Pediatrics, University of Messina, Via Terranova 9, Messina, Italy.
J Electrocardiol. 2009 Nov-Dec;42(6):633-5. doi: 10.1016/j.jelectrocard.2009.03.013. Epub 2009 Apr 18.
In a patient with atrial tachycardia with a rate of 200 per minute, the A-V conduction ratio was at times 2:1, but often it was 4:3 or 3:2 with progressive P-R interval prolongation (Wenckebach mechanism, an expression of presumable A-V nodal block). In each episode of 4:3 conduction, the first QRS complex was narrow, and the 2 ensuing beats were wide due to aberrant conduction. Aberration did not occur with a constant configuration, but in consecutive episodes of 4:3 conduction ratio there was a regular alternation of left bundle branch block and right bundle branch block. The pattern was explained by concealed retrograde conduction into the anterogradely blocked bundle branch. This caused 2 distinct effects: (1) shifting "to the right" of the refractory period of the affected bundle branch, resulting in maintenance of aberration with the same configuration, if consecutive atrial impulses were conducted to the ventricles, and (2) shortening of the effective cycle of the affected bundle branch, resulting in aberration due to block of the controlateral bundle branch, whenever a pause occasioned by a nonconducted atrial impulse was followed by restoration of 1:1 conduction for 2 or more consecutive beats.
在一名房性心动过速且心率为每分钟200次的患者中,房室传导比例有时为2:1,但常为4:3或3:2,并伴有进行性P-R间期延长(文氏机制,提示可能存在房室结阻滞)。在每一次4:3传导的发作中,第一个QRS波群窄,随后的2个搏动宽,系因差异性传导所致。差异性传导并非以固定形态出现,而是在连续的4:3传导发作中,左束支阻滞和右束支阻滞呈规律交替。这种模式是由隐匿性逆行传导至前向阻滞的束支来解释的。这产生了2种不同的效应:(1)使受影响束支的不应期“右移”,如果连续的心房冲动传导至心室,则导致相同形态的差异性传导持续存在;(2)缩短受影响束支的有效周期,每当未传导的心房冲动引起的间歇之后连续2次或更多次搏动恢复1:1传导时,因对侧束支阻滞而导致差异性传导。