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起源于主动脉无冠窦的房性心动过速引发的心房颤动成功消融。

Atrial tachycardia initiating atrial fibrillation successfully ablated in the non-coronary cusp of the aorta.

作者信息

Yamada Takumi, Allison J Scott, McElderry H Thomas, Doppalapudi Harish, Kay G Neal

机构信息

Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL 35294-0019, USA.

出版信息

J Interv Card Electrophysiol. 2010 Mar;27(2):123-6. doi: 10.1007/s10840-009-9396-5. Epub 2009 Apr 18.

Abstract

A 60-year-old woman was referred for catheter ablation of atrial fibrillation (AF). Atrial flutter and atrial tachycardia (AT) also had been clinically documented. During the electrophysiological study, the clinical AT was induced by burst atrial pacing during isoproterenol infusion and exhibited negative P waves in the inferior leads, positive P waves in leads I, aVL, and aVR, and biphasic P waves in lead V1. The AT repeatedly and spontaneously accelerated to initiate AF by causing fibrillatory conduction in the atria. Successful catheter ablation of the AT was achieved in the non-coronary cusp of the aorta (NCC) where the local atrio-ventricular electrogram amplitude ratio was >1 during both the AT and sinus rhythm. The tailored approach targeting the NCC AT alone without left atrial ablation completely eliminated the AF. In catheter ablation of AF in a patient with a co-existing clinical AT, it may be recommended to examine the clinical AT first. If the clinical AT initiates the AF and local atrial activations in the His bundle region precede the P wave onset during AT, mapping in the NCC should be considered prior to left atrial catheterization.

摘要

一名60岁女性因房颤(AF)接受导管消融治疗。临床上也记录到了房扑和房性心动过速(AT)。在电生理研究期间,临床AT在输注异丙肾上腺素期间通过短阵房性起搏诱发,下壁导联出现负向P波,I、aVL和aVR导联出现正向P波,V1导联出现双向P波。AT反复自发加速,通过在心房引起颤动传导引发房颤。在主动脉无冠窦(NCC)成功进行了AT导管消融,在AT和窦性心律期间,该部位局部房室电图振幅比均>1。仅针对NCC AT而不进行左心房消融的定制方法完全消除了房颤。在合并临床AT的患者进行房颤导管消融时,可能建议首先检查临床AT。如果临床AT引发房颤,且在AT期间希氏束区域的局部心房激动先于P波起始,则在进行左心房导管插入术前应考虑在NCC进行标测。

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