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本文引用的文献
1
Defective homing is associated with altered Cdc42 activity in cells from patients with Fanconi anemia group A.
Blood. 2008 Sep 1;112(5):1683-6. doi: 10.1182/blood-2008-03-147090. Epub 2008 Jun 18.
2
AMD3100 synergizes with G-CSF to mobilize repopulating stem cells in Fanconi anemia knockout mice.
Exp Hematol. 2008 Sep;36(9):1084-90. doi: 10.1016/j.exphem.2008.03.016. Epub 2008 May 20.
3
CXCR4 induction in hematopoietic progenitor cells from Fanca(-/-), -c(-/-), and -d2(-/-) mice.
Exp Hematol. 2008 Mar;36(3):273-82. doi: 10.1016/j.exphem.2007.11.006.
4
Rho GTPase Cdc42 coordinates hematopoietic stem cell quiescence and niche interaction in the bone marrow.
Proc Natl Acad Sci U S A. 2007 Mar 20;104(12):5091-6. doi: 10.1073/pnas.0610819104. Epub 2007 Mar 12.
5
Hematopoietic stem cells from fancc(-/-) mice have lower growth and differentiation potential in response to growth factors.
Stem Cells. 2002;20(5):438-47. doi: 10.1634/stemcells.20-5-438.
6
The Fanconi anemia group C gene product: signaling functions in hematopoietic cells.
Exp Hematol. 2001 Dec;29(12):1371-81. doi: 10.1016/s0301-472x(01)00755-x.
7
Rapid and efficient homing of human CD34(+)CD38(-/low)CXCR4(+) stem and progenitor cells to the bone marrow and spleen of NOD/SCID and NOD/SCID/B2m(null) mice.
Blood. 2001 May 15;97(10):3283-91. doi: 10.1182/blood.v97.10.3283.
8
Activation of the small GTPase Cdc42 by the inflammatory cytokines TNF(alpha) and IL-1, and by the Epstein-Barr virus transforming protein LMP1.
J Cell Sci. 1999 Sep;112 ( Pt 17):2983-92. doi: 10.1242/jcs.112.17.2983.
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