Gopinathannair Rakesh, Sandesara Chirag M, Olshansky Brian
Division of Cardiovascular Medicine, University of Iowa Hospitals and Clinics, E 318-5 GH, 200 Hawkins Drive, Iowa City, IA 52242, USA.
Europace. 2009 Sep;11(9):1230-4. doi: 10.1093/europace/eup154. Epub 2009 Jun 18.
A patient with atrial flutter, intermittent non-sustained wide complex tachycardia and 1:1 AV conduction is reported. Electrophysiology testing showed counterclockwise isthmus-dependent right atrial flutter with conduction via the AV node and an innocent bystander left lateral accessory pathway. This explained the observed intermittent wide complex tachycardia. After successful bidirectional cavotricuspid isthmus conduction block, a sustained wide complex tachycardia with identical counterclockwise right atrial activation and rate occurred. This was due to antidromic AV re-entrant tachycardia with innocent bystander activation of the right atrium mimicking atrial flutter. Accessory pathway ablation effectively stopped tachycardia.
报道了一名患有心房扑动、间歇性非持续性宽QRS心动过速及1:1房室传导的患者。电生理检查显示为逆时针方向峡部依赖性右房扑动,通过房室结传导,并有一条无临床意义的旁观者左外侧旁路。这解释了观察到的间歇性宽QRS心动过速。成功进行双向腔静脉三尖瓣峡部传导阻滞之后,出现了持续的宽QRS心动过速,其右房激动顺序和频率与之前相同。这是由于逆向房室折返性心动过速,右房被无临床意义的旁观者激动,类似心房扑动。消融旁路有效地终止了心动过速。