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在组成性表达人嗜T淋巴细胞病毒I型(HTLV-I)Tax蛋白的细胞中,对激活转录因子NF-κB所需信号的抑制作用。

Suppression of signals required for activation of transcription factor NF-kappa B in cells constitutively expressing the HTLV-I Tax protein.

作者信息

Ruben S M, Rosen C A

机构信息

Department of Molecular Oncology and Virology, Roche Institute of Molecular Biology, Roche Research Center, Nutley, NJ 07110.

出版信息

New Biol. 1990 Oct;2(10):894-902.

PMID:1964088
Abstract

Transient short-term expression of the Tax protein of human T-cell leukemia virus type-I (HTLV-I) leads to activation of the pleiotropic transcription factor NF-kappa B. Consistent with findings obtained with transient expression assays, we observed marked accumulation of the transcription factor NF-kappa B in the nucleus of Namalwa B lymphoid cells, which constitutively express Tax. In contrast, NF-kappa B activity was not detected in the nucleus following long-term expression of Tax in Jurkat T lymphocytes. The ability of both mitogens and cytokines to activate NF-kappa B was also blocked in Jurkat cells constitutively expressing Tax. However, the activation of other mitogen-inducible transcription factors, such as Fos and Jun, was unaffected. Thus, depending on the cellular environment, the short- and long-term effects of Tax expression can be quite different. Consequently, one function of Tax in cells infected with HTLV-I might involve cell-type-specific suppression, as opposed to activation, of distinct signal pathways. The cells lines described here should be useful for the delineation of signaling pathways utilized in the selective regulation of gene expression.

摘要

人T细胞白血病病毒I型(HTLV-I)的Tax蛋白的瞬时短期表达会导致多效转录因子NF-κB的激活。与瞬时表达分析的结果一致,我们观察到在组成性表达Tax的Namalwa B淋巴细胞的细胞核中,转录因子NF-κB有明显积累。相比之下,在Jurkat T淋巴细胞中长期表达Tax后,未在细胞核中检测到NF-κB活性。在组成性表达Tax的Jurkat细胞中,丝裂原和细胞因子激活NF-κB的能力也受到了阻碍。然而,其他丝裂原诱导型转录因子,如Fos和Jun的激活并未受到影响。因此,根据细胞环境的不同,Tax表达的短期和长期效应可能会有很大差异。因此,Tax在感染HTLV-I的细胞中的一个功能可能涉及对不同信号通路的细胞类型特异性抑制,而不是激活。这里描述的细胞系对于描绘在基因表达的选择性调控中所利用的信号通路应该是有用的。

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