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本文引用的文献
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Cullin mediates degradation of RhoA through evolutionarily conserved BTB adaptors to control actin cytoskeleton structure and cell movement.
Mol Cell. 2009 Sep 24;35(6):841-55. doi: 10.1016/j.molcel.2009.09.004.
2
Cul3 overexpression depletes Nrf2 in breast cancer and is associated with sensitivity to carcinogens, to oxidative stress, and to chemotherapy.
Mol Cancer Ther. 2009 Aug;8(8):2432-40. doi: 10.1158/1535-7163.MCT-08-1186. Epub 2009 Jul 28.
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An inhibitor of NEDD8-activating enzyme as a new approach to treat cancer.
Nature. 2009 Apr 9;458(7239):732-6. doi: 10.1038/nature07884.
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Suprafacial orientation of the SCFCdc4 dimer accommodates multiple geometries for substrate ubiquitination.
Cell. 2007 Jun 15;129(6):1165-76. doi: 10.1016/j.cell.2007.04.042.
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A Cul3-based E3 ligase removes Aurora B from mitotic chromosomes, regulating mitotic progression and completion of cytokinesis in human cells.
Dev Cell. 2007 Jun;12(6):887-900. doi: 10.1016/j.devcel.2007.03.019.
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Rho GTPases: biochemistry and biology.
Annu Rev Cell Dev Biol. 2005;21:247-69. doi: 10.1146/annurev.cellbio.21.020604.150721.
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Sequence and structural analysis of BTB domain proteins.
Genome Biol. 2005;6(10):R82. doi: 10.1186/gb-2005-6-10-r82. Epub 2005 Sep 15.
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Molecular mechanisms activating the Nrf2-Keap1 pathway of antioxidant gene regulation.
Antioxid Redox Signal. 2005 Mar-Apr;7(3-4):385-94. doi: 10.1089/ars.2005.7.385.
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