Jastrzebski Marek
I Klinika Kardiologii i Nadciśnienia Tetniczego, Szpital Uniwersytecki, Kraków.
Kardiol Pol. 2009 Sep;67(9):1030-5 discussion 1035-6.
Three cases of possible supernormal conduction and excitability are described. Case one: subthreshold stimulation with ventricular capture occurring only at the end of the T wave. Case two: advanced atrioventricular block with conduction present only when P waves fell at the downsloping part of the T wave of ventricular escape beats. Case three: 2 : 1 atrioventricular block at atrial pacing rate of 115 bpm with improvement of conduction during higher pacing rates (3 : 2 block at 130 bpm and above; with negligible PR prolongation). Possible alternative mechanisms: gap phenomenon, ventriculophasic modulation of vagal discharge, ventriculophasic pacing lead movement and concealed 2 : 1 atrioventricular block due to superimposed ventricular escape rhythm, are discussed and considered either impossible or unlikely.
描述了三例可能的超常传导和兴奋性病例。病例一:阈下刺激,仅在T波末端出现心室夺获。病例二:高度房室传导阻滞,仅当P波落在心室逸搏T波的下降支时才有传导。病例三:心房起搏频率为115次/分时呈2∶1房室传导阻滞,较高起搏频率时(130次/分及以上时呈3∶2阻滞;PR间期延长可忽略不计)传导改善。讨论了可能的其他机制:缝隙现象、迷走神经放电的心室相性调制、心室相性起搏导线移动以及由于叠加心室逸搏心律导致的隐匿性2∶1房室传导阻滞,并认为这些机制要么不可能,要么不太可能。