SUNY Upstate Medical University, 750 E. Adams Street, Syracuse, NY 13210, USA.
Foot Ankle Int. 2010 Apr;31(4):320-8. doi: 10.3113/FAI.2010.0320.
Tibialis posterior muscle weakness has been documented in subjects with Stage II posterior tibial tendon dysfunction (PTTD) but the effect of weakness on foot structure remains unclear. The association between strength and flatfoot kinematics may guide treatment such as the use of strengthening programs targeting the tibialis posterior muscle.
Thirty Stage II PTTD subjects (age; 58.1 +/- 10.5 years, BMI 30.6 +/- 5.4) and 15 matched controls (age; 56.5 +/- 7.7 years, BMI 30.6 +/- 3.6) volunteered for this study. Deep Posterior Compartment strength was measured from both legs of each subject and the strength ratio was used to compare each subject's involved side to their uninvolved side. A 20% deficit was defined, a priori, to define two groups of subjects with PTTD. The strength ratio for each group averaged; 1.06 +/- 0.1 (range 0.87 to 1.36) for controls, 1.06 +/- 0.1 (range, 0.89 to 1.25), for the PTTD strong group, and 0.64 +/- 0.2 (range 0.42 to 0.76) for the PTTD weak group. Across four phases of stance, kinematic measures of flatfoot were compared between the three groups using a two-way mixed effect ANOVA model repeated for each kinematic variable.
Subjects with PTTD regardless of group demonstrated significantly greater hindfoot eversion compared to controls. Subjects with PTTD who were weak demonstrated greater hindfoot eversion compared to subjects with PTTD who were strong. For forefoot abduction and MLA angles the differences between groups depended on the phase of stance with significant differences between each group observed at the pre-swing phase of stance.
Strength was associated with the degree of flatfoot deformity observed during walking, however, flatfoot deformity may also occur without strength deficits.
Strengthening programs may only partially correct flatfoot kinematics while other clinical interventions such as bracing or surgery may also be indicated.
胫骨后肌力量减弱已在 II 期后胫骨后肌腱功能障碍(PTTD)患者中得到证实,但力量对足结构的影响尚不清楚。力量与扁平足运动学之间的关联可能指导治疗,例如使用针对胫骨后肌的强化计划。
30 名 II 期 PTTD 受试者(年龄 58.1 ± 10.5 岁,BMI 30.6 ± 5.4)和 15 名匹配的对照者(年龄 56.5 ± 7.7 岁,BMI 30.6 ± 3.6)自愿参加了这项研究。从每位受试者的双腿测量深部后间隔力量,并使用力量比来比较每位受试者的受累侧与非受累侧。先前定义了 20%的缺陷,以定义两组 PTTD 患者。每组的力量比平均为:对照组为 1.06 ± 0.1(范围为 0.87 至 1.36),PTTD 强组为 1.06 ± 0.1(范围为 0.89 至 1.25),PTTD 弱组为 0.64 ± 0.2(范围为 0.42 至 0.76)。在四个站立阶段,使用双向混合效应 ANOVA 模型重复每个运动学变量,比较三组之间扁平足的运动学测量值。
无论组如何,PTTD 患者的后足外翻明显大于对照组。与 PTTD 强组相比,PTTD 弱组的后足外翻更大。对于前足外展和 MLA 角度,组间差异取决于站立阶段,在站立预摆动阶段观察到每组之间存在显著差异。
力量与行走时观察到的扁平足畸形程度相关,但即使没有力量缺陷,扁平足畸形也可能发生。
强化计划可能只能部分纠正扁平足运动学,而其他临床干预措施,如支具或手术,也可能是必要的。