Sex modulates intestinal transformation by the tumor-suppressor GCC.

作者信息

Li Peng, Schulz Stephanie, Pitari Giovanni M, Waldman Scott A

机构信息

Department of Pharmacology and Experimental Therapeutics, Thomas Jefferson University, Philadelphia, Pennsylvania, USA.

出版信息

Clin Transl Sci. 2008 Sep;1(2):150. doi: 10.1111/j.1752-8062.2008.00010.x.

DOI:10.1111/j.1752-8062.2008.00010.x

PMID:20443838

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC3137371/

Abstract

摘要

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Sex modulates intestinal transformation by the tumor-suppressor GCC.

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[8]

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[10]

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Homeostatic control of the crypt-villus axis by the bacterial enterotoxin receptor guanylyl cyclase C restricts the proliferating compartment in intestine.

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Clin Pharmacol Ther. 2007-10

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Guanylyl cyclase C suppresses intestinal tumorigenesis by restricting proliferation and maintaining genomic integrity.

Gastroenterology. 2007-8

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Differences between right- and left-sided colon cancer in patient characteristics, cancer morphology and histology.

J Gastroenterol Hepatol. 2008-3

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Role of ERbeta palmitoylation in the inhibition of human colon cancer cell proliferation.

Endocr Relat Cancer. 2007-3

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Estrogen receptors alpha and beta are inhibitory modifiers of Apc-dependent tumorigenesis in the proximal colon of Min/+ mice.

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A validated quantitative assay to detect occult micrometastases by reverse transcriptase-polymerase chain reaction of guanylyl cyclase C in patients with colorectal cancer.

Clin Cancer Res. 2006-8-1

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CpG island methylator phenotype underlies sporadic microsatellite instability and is tightly associated with BRAF mutation in colorectal cancer.

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Clinicopathological characteristics, microsatellite instability, and expression of mucin core proteins and p53 in colorectal mucinous adenocarcinomas in relation to location.

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Sex modulates intestinal transformation by the tumor-suppressor GCC.

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