Adult testicular dysgenesis of Inhba conditional knockout mice may also be caused by disruption of cross-talk between Leydig cells and germ cells.
作者信息
Sun Zhijian, Li Zhen, Zhang Yuanqiang
出版信息
Proc Natl Acad Sci U S A. 2010 Aug 31;107(35):E135; author reply E136. doi: 10.1073/pnas.1008771107. Epub 2010 Aug 5.
Abstract
摘要
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Adult testicular dysgenesis of Inhba conditional knockout mice may also be caused by disruption of cross-talk between Leydig cells and germ cells.
Proc Natl Acad Sci U S A. 2010 Aug 31;107(35):E135; author reply E136. doi: 10.1073/pnas.1008771107. Epub 2010 Aug 5.
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本文引用的文献
1
Activin A, a product of fetal Leydig cells, is a unique paracrine regulator of Sertoli cell proliferation and fetal testis cord expansion.
Proc Natl Acad Sci U S A. 2010 Jun 8;107(23):10526-31. doi: 10.1073/pnas.1000318107. Epub 2010 May 24.
2
Activin bioactivity affects germ cell differentiation in the postnatal mouse testis in vivo.
Biol Reprod. 2010 May;82(5):980-90. doi: 10.1095/biolreprod.109.079855. Epub 2010 Feb 3.
3
Inhibin, activin, follistatin and FSH serum levels and testicular production are highly modulated during the first spermatogenic wave in mice.
Reproduction. 2008 Sep;136(3):345-59. doi: 10.1530/REP-08-0140. Epub 2008 May 30.
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Detection of anti-Mullerian hormone receptor II protein in the postnatal rat testis from birth to sexual maturity.
Histol Histopathol. 2006 Feb;21(2):125-30. doi: 10.14670/HH-21.125.
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Regulation of germ cell and Sertoli cell development by activin, follistatin, and FSH.
Dev Biol. 2000 Apr 15;220(2):225-37. doi: 10.1006/dbio.2000.9625.
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