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巴西副球孢子菌 GP43 衍生肽是局部和全身炎症反应的有效调节剂。

Paracoccidioides brasiliensis GP43-derived peptides are potent modulators of local and systemic inflammatory response.

机构信息

Universidade Federal de São Paulo - UNIFESP, Department of Microbiology, Immunology and Parasitology, Discipline of Immunology, São Paulo, Brazil.

出版信息

Microbes Infect. 2012 Jun;14(6):517-27. doi: 10.1016/j.micinf.2011.12.012. Epub 2012 Jan 10.

Abstract

Paracoccidioidomycosis is a systemic granulomatous disease caused by the dimorphic fungus Paracoccidioides brasiliensis. Its major antigen is a 43 kDa glycoprotein whose peptides embody different functions: P10 peptide, a T-cell epitope, induces protective response while P4 and P23 peptides inhibit both, macrophage functions and inflammatory reaction, thus facilitating infection. Here we investigated the modulating mechanisms of the immune response exerted by P4 and P23 involved in the latter inhibitory effect on macrophages. Moreover we analyzed the peptides effects in different models in vivo. While evaluating whether P4 and P23 present systemic anti-inflammatory effects in vivo, we showed that their intraperitonial administration decreased footpad swelling in mice infected with either P. brasiliensis or Mycobacterium bovis. Both, qPCR and ELISA assays suggested that this anti-inflammatory effect depended on alterations in the kinetics of production of innate immunity modulators such as TNF-α, IL6, IL10 and TLR2. IL10 seems to be early produced than TNF-α and IL6, produced later in presence of peptides. Higher doses or intravenously given P4 and P23 resulted in earlier and more prolonged anti-inflammatory effects. Moreover, continuous treatment with P4 and P23 sustained the anti-inflammatory activity throughout.

摘要

巴西副球孢子菌病是一种由双相真菌巴西副球孢子菌引起的系统性肉芽肿疾病。其主要抗原是一种 43 kDa 的糖蛋白,其肽段具有不同的功能:P10 肽是 T 细胞表位,可诱导保护性反应,而 P4 和 P23 肽则抑制巨噬细胞的功能和炎症反应,从而促进感染。在这里,我们研究了 P4 和 P23 对巨噬细胞的后者抑制作用所发挥的免疫反应调节机制。此外,我们还分析了这些肽在不同体内模型中的作用。在评估 P4 和 P23 是否在体内具有全身抗炎作用时,我们发现它们腹腔内给药可减少感染巴西副球孢子菌或牛分枝杆菌的小鼠的足垫肿胀。qPCR 和 ELISA 检测均表明,这种抗炎作用取决于先天免疫调节剂如 TNF-α、IL6、IL10 和 TLR2 产生的动力学变化。IL10 似乎比 TNF-α和 IL6 更早产生,在存在肽的情况下产生得更晚。更高剂量或静脉内给予 P4 和 P23 导致更早和更持久的抗炎作用。此外,连续给予 P4 和 P23 可维持整个抗炎活性。

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