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本文引用的文献
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Global patterns of tissue-specific alternative polyadenylation in Drosophila.
Cell Rep. 2012 Mar 29;1(3):277-89. doi: 10.1016/j.celrep.2012.01.001.
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The poly(A)-binding protein nuclear 1 suppresses alternative cleavage and polyadenylation sites.
Cell. 2012 Apr 27;149(3):538-53. doi: 10.1016/j.cell.2012.03.022. Epub 2012 Apr 12.
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A Pre-mRNA degradation pathway that selectively targets intron-containing genes requires the nuclear poly(A)-binding protein.
Mol Cell. 2011 Oct 7;44(1):108-19. doi: 10.1016/j.molcel.2011.06.035.
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Mechanisms and consequences of alternative polyadenylation.
Mol Cell. 2011 Sep 16;43(6):853-66. doi: 10.1016/j.molcel.2011.08.017.
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Ending the message: poly(A) signals then and now.
Genes Dev. 2011 Sep 1;25(17):1770-82. doi: 10.1101/gad.17268411.
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The nuclear poly(A)-binding protein interacts with the exosome to promote synthesis of noncoding small nucleolar RNAs.
Mol Cell. 2010 Jan 15;37(1):34-45. doi: 10.1016/j.molcel.2009.12.019.
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Poly(A) tail length is controlled by the nuclear poly(A)-binding protein regulating the interaction between poly(A) polymerase and the cleavage and polyadenylation specificity factor.
J Biol Chem. 2009 Aug 21;284(34):22803-14. doi: 10.1074/jbc.M109.018226. Epub 2009 Jun 9.
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An essential cytoplasmic function for the nuclear poly(A) binding protein, PABP2, in poly(A) tail length control and early development in Drosophila.
Dev Cell. 2005 Oct;9(4):511-22. doi: 10.1016/j.devcel.2005.09.002.
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Stimulation of poly(A) polymerase through a direct interaction with the nuclear poly(A) binding protein allosterically regulated by RNA.
EMBO J. 2003 Jul 15;22(14):3705-14. doi: 10.1093/emboj/cdg347.
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Tissue-specific autoregulation of Drosophila suppressor of forked by alternative poly(A) site utilization leads to accumulation of the suppressor of forked protein in mitotically active cells.
RNA. 2000 Nov;6(11):1529-38. doi: 10.1017/s1355838200001266.
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