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本文引用的文献
1
Exonuclease hDIS3L2 specifies an exosome-independent 3'-5' degradation pathway of human cytoplasmic mRNA.
EMBO J. 2013 Jul 3;32(13):1855-68. doi: 10.1038/emboj.2013.135. Epub 2013 Jun 11.
2
A role for the Perlman syndrome exonuclease Dis3l2 in the Lin28-let-7 pathway.
Nature. 2013 May 9;497(7448):244-8. doi: 10.1038/nature12119. Epub 2013 Apr 17.
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Lin28: primal regulator of growth and metabolism in stem cells.
Cell Stem Cell. 2013 Apr 4;12(4):395-406. doi: 10.1016/j.stem.2013.03.005.
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The exoribonuclease Dis3L2 defines a novel eukaryotic RNA degradation pathway.
EMBO J. 2013 Jul 3;32(13):1842-54. doi: 10.1038/emboj.2013.63. Epub 2013 Mar 15.
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Regulation of cytoplasmic mRNA decay.
Nat Rev Genet. 2012 Mar 6;13(4):246-59. doi: 10.1038/nrg3160.
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Germline mutations in DIS3L2 cause the Perlman syndrome of overgrowth and Wilms tumor susceptibility.
Nat Genet. 2012 Feb 5;44(3):277-84. doi: 10.1038/ng.1071.
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Conserved RNaseII domain protein functions in cytoplasmic mRNA decay and suppresses Arabidopsis decapping mutant phenotypes.
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TUT4 in concert with Lin28 suppresses microRNA biogenesis through pre-microRNA uridylation.
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Decapping is preceded by 3' uridylation in a novel pathway of bulk mRNA turnover.
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Degradation of histone mRNA requires oligouridylation followed by decapping and simultaneous degradation of the mRNA both 5' to 3' and 3' to 5'.
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