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对神经血管龛进行建模:对缺氧损伤的小鼠侧脑室下区进行无偏转录组分析。

Modeling the neurovascular niche: unbiased transcriptome analysis of the murine subventricular zone in response to hypoxic insult.

机构信息

Department of Pathology, Yale University School of Medicine, New Haven, Connecticut, United States of America.

出版信息

PLoS One. 2013 Oct 11;8(10):e76265. doi: 10.1371/journal.pone.0076265. eCollection 2013.

Abstract

Premature infants often experience chronic hypoxia, resulting in cognitive & motor neurodevelopmental handicaps. These sometimes devastating handicaps are thought to be caused by compromised neural precursor cell (NPC) repair/recovery resulting in variable central nervous system (CNS) repair/recovery. We have identified differential responses of two mouse strains (C57BL/6 & CD1) to chronic hypoxia that span the range of responsiveness noted in the premature human population. We previously correlated several CNS tissue and cellular behaviors with the different behavioral parameters manifested by these two strains. In this report, we use unbiased array technology to interrogate the transcriptome of the subventricular zone (SVZ) in these strains. Our results illustrate differences in mRNA expression in the SVZ of both C57BL/6 and CD1 mice following hypoxia as well as differences between C57BL/6 and CD1 SVZ under both normoxic and hypoxic conditions. Differences in expression were found in gene sets associated with Sox10-mediated neural functions that explain, in part, the differential cognitive and motor responsiveness to hypoxic insult. This may shed additional light on our understanding of the variable responses noted in the human premature infant population and facilitate early intervention approaches. Further interrogation of the differentially expressed gene sets will provide a more complete understanding of the differential responses to, and recovery from, hypoxic insult allowing for more informed modeling of the ranges of disease severity observed in the very premature human population.

摘要

早产儿经常经历慢性缺氧,导致认知和运动神经发育障碍。这些有时是毁灭性的障碍被认为是由于神经前体细胞 (NPC) 的修复/恢复受损,导致中枢神经系统 (CNS) 的修复/恢复情况不同。我们已经确定了两种小鼠品系 (C57BL/6 和 CD1) 对慢性缺氧的不同反应,这些反应涵盖了早产儿人群中观察到的反应范围。我们之前将几种中枢神经系统组织和细胞行为与这两种品系表现出的不同行为参数相关联。在本报告中,我们使用无偏置阵列技术来研究这两种品系的侧脑室下区 (SVZ) 的转录组。我们的结果表明,在缺氧后,C57BL/6 和 CD1 小鼠的 SVZ 中的 mRNA 表达存在差异,以及在常氧和缺氧条件下,C57BL/6 和 CD1 SVZ 之间的差异。在与 Sox10 介导的神经功能相关的基因集的表达存在差异,这些差异部分解释了对缺氧损伤的认知和运动反应的差异。这可能进一步阐明我们对人类早产儿人群中观察到的可变反应的理解,并促进早期干预方法。对差异表达基因集的进一步研究将提供对缺氧损伤的反应和恢复的更全面理解,从而更明智地对非常早产儿人群中观察到的疾病严重程度范围进行建模。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2325/3795763/7ce49ccc5ead/pone.0076265.g001.jpg

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