Yeh S J, Fu M, Lin F C, Chang C H, Hung J S
J Electrocardiol. 1986 Apr;19(2):193-6. doi: 10.1016/s0022-0736(86)80028-0.
We report a unique patient in whom electrophysiologic studies elucidated the mechanism of a rare form of swallowing-induced atrioventricular reentrant tachycardia, and for whom successful surgical ablation of an accessory pathway abolished intractable episodes of palpitation. A 64-year-old man was incapacitated by frequent attacks of palpitation following swallowing. Electrocardiograms documented paroxysmal supraventricular tachycardias initiated by a premature atrial beat or beats following swallowing. During electrophysiologic studies swallowing consistently induced premature atrial beats which in turn initiated a sustained atrioventricular reentrant tachycardia incorporating a retrogradely conducting left-sided concealed accessory pathway. The atrial activation sequence related to the premature atrial beats and the morphology of the premature P waves suggested that premature atrial beats originated in the right atrium. The mechanism of induction of premature atrial beats following swallowing remains obscure in our patient. Antiarrythmic drugs failed to prevent induction of sustained tachycardias during sequential electrophysiologic studies. The patient underwent successful surgical ablation of the accessory pathway and is free from palpitation 15 months after the surgery.
我们报告了一位独特的患者,电生理研究阐明了一种罕见的吞咽诱发房室折返性心动过速的机制,并且成功地通过手术消融旁路消除了该患者的顽固性心悸发作。一名64岁男性因吞咽后频繁发作心悸而丧失活动能力。心电图记录显示,吞咽后由房性早搏引发阵发性室上性心动过速。在电生理研究期间,吞咽持续诱发房性早搏,进而引发持续的房室折返性心动过速,其中包含一条逆向传导的左侧隐匿性旁路。与房性早搏相关的心房激动顺序以及早搏P波的形态表明,房性早搏起源于右心房。在我们的患者中,吞咽后诱发房性早搏的机制仍不清楚。在系列电生理研究中,抗心律失常药物未能预防持续性心动过速的诱发。该患者成功接受了旁路手术消融,术后15个月未再出现心悸。