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染料木黄酮对雄性B6C3F1小鼠链脲佐菌素糖尿病的调节作用可通过饮食诱导。

Genistein modulation of streptozotocin diabetes in male B6C3F1 mice can be induced by diet.

作者信息

Guo Tai L, Wang Yunbiao, Xiong Tao, Ling Xiao, Zheng Jianfeng

机构信息

Department of Biosciences and Diagnostic Imaging, College of Veterinary Medicine, University of Georgia, Athens, GA 30602-7382, USA.

Department of Biosciences and Diagnostic Imaging, College of Veterinary Medicine, University of Georgia, Athens, GA 30602-7382, USA; Key Laboratory of Wetland Ecology and Environment, Northeast Institute of Geography and Agroecology, Chinese Academy of Sciences, Changchun 130102, China.

出版信息

Toxicol Appl Pharmacol. 2014 Nov 1;280(3):455-66. doi: 10.1016/j.taap.2014.08.028. Epub 2014 Aug 30.

Abstract

Diet and phytoestrogens affect the development and progression of diabetes. The objective of the present study was to determine if oral exposure to phytoestrogen genistein (GE) by gavage changed blood glucose levels (BGL) through immunomodulation in streptozotocin (STZ)-induced diabetic male B6C3F1 mice fed with three different diets. These three diets were: NTP-2000 diet (NTP), soy- and alfalfa-free 5K96 diet (SOF) and high fat diet (HFD) with 60% of kcal from fat, primarily rendered fat of swine. The dosing regimen for STZ consisted of three 100mg/kg doses (i.p.): the first dose was administered at approximately 2weeks following the initiation of daily GE (20mg/kg) gavage, and the second dose was on day 19 following the first dose, and the third dose was on day 57 following the first dose. In mice on the NTP diet, GE treatment decreased BGL with statistical significances observed on days 33 and 82 following the first STZ injection. In mice fed the HFD diet, GE treatment produced a significant decrease and a significant increase in BGL on days 15 and 89 following the first STZ injection, respectively. In mice fed the SOF diet, GE treatment had no significant effects on BGL. Although GE treatment affected phenotypic distributions of both splenocytes (T cells, B cells, natural killer cells and neutrophils) and thymocytes (CD4/CD8 and CD44/CD25), and their mitochondrial transmembrane potential and generation of reactive oxygen species, indicators of cell death (possibly apoptosis), GE modulation of neutrophils was more consistent with its diabetogenic or anti-diabetic potentials. The differential effects of GE on BGL in male B6C3F1 mice fed with three different diets with varied phytoestrogen contents suggest that the estrogenic properties of this compound may contribute to its modulation of diabetes.

摘要

饮食和植物雌激素会影响糖尿病的发展和进程。本研究的目的是确定通过灌胃口服植物雌激素染料木黄酮(GE)是否会通过免疫调节作用改变链脲佐菌素(STZ)诱导的糖尿病雄性B6C3F1小鼠的血糖水平(BGL),这些小鼠分别喂食三种不同的饮食。这三种饮食分别是:NTP - 2000饮食(NTP)、不含大豆和苜蓿的5K96饮食(SOF)以及高脂肪饮食(HFD),其中60%的热量来自脂肪,主要是猪脂肪。STZ的给药方案包括三次腹腔注射,剂量为100mg/kg:第一剂在开始每日GE(20mg/kg)灌胃后约2周给药,第二剂在第一剂给药后的第19天给药,第三剂在第一剂给药后的第57天给药。在食用NTP饮食的小鼠中,GE治疗降低了BGL,在首次注射STZ后的第33天和第82天观察到统计学意义。在食用HFD饮食的小鼠中,GE治疗在首次注射STZ后的第15天和第89天分别使BGL显著降低和显著升高。在食用SOF饮食的小鼠中,GE治疗对BGL没有显著影响。尽管GE治疗影响了脾细胞(T细胞、B细胞、自然杀伤细胞和中性粒细胞)和胸腺细胞(CD4/CD8和CD44/CD25)的表型分布,以及它们的线粒体跨膜电位和活性氧的产生,这些都是细胞死亡(可能是凋亡)的指标,但GE对中性粒细胞的调节与其致糖尿病或抗糖尿病潜力更为一致。GE对喂食三种不同植物雌激素含量饮食的雄性B6C3F1小鼠BGL的不同影响表明,该化合物的雌激素特性可能有助于其对糖尿病的调节作用。

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