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γH2AX 病灶出现在看似完整的有丝分裂染色体上:并非错配修复事件的特征,而是未解决的 DNA 损伤信号。

γH2AX foci on apparently intact mitotic chromosomes: not signatures of misrejoining events but signals of unresolved DNA damage.

作者信息

Martín Marta, Terradas Mariona, Hernández Laia, Genescà Anna

机构信息

a Department of Cell Biology, Immunology, and Neuroscience ; Autonomous University of Barcelona ; Barcelona , Spain.

出版信息

Cell Cycle. 2014;13(19):3026-36. doi: 10.4161/15384101.2014.947786.

Abstract

The presence of γH2AX foci on apparently intact mitotic chromosomes is controversial because they challenge the assumed relationship between γH2AX foci and DNA double-strand breaks (DSBs). In this work, we show that after irradiation during interphase, a variety of γH2AX foci are scored in mitotic cells. Surprisingly, approximately 80% of the γH2AX foci spread over apparently undamaged chromatin at Terminal or Interstitial positions and they can display variable sizes, thus being classified as Small, Medium and Big foci. Chromosome and chromatid breaks that reach mitosis are spotted with Big (60%) and Medium (30%) Terminal γH2AX foci, but very rarely are they signaled with Small γH2AX foci. To evaluate if Interstitial γH2AX foci might be signatures of misrejoining, an mFISH analysis was performed on the same slides. The results show that Interstitial γH2AX foci lying on apparently intact chromatin do not mark sites of misrejoining, and that misrejoined events were never signaled by a γH2AX foci during mitosis. Finally, when analyzing the presence of other DNA-damage response (DDR) factors we found that all γH2AX foci-regardless their coincidence with a visible break-always colocalized with MRE11, but not with 53BP1. This pattern suggests that these γH2AX foci may be hallmarks of both microscopically visible and invisible DNA damage, in which an active, although incomplete or halted DDR is taking place.

摘要

γH2AX焦点出现在看似完整的有丝分裂染色体上这一现象存在争议,因为它们对γH2AX焦点与DNA双链断裂(DSB)之间的假定关系提出了挑战。在这项研究中,我们发现间期照射后,有丝分裂细胞中可检测到多种γH2AX焦点。令人惊讶的是,大约80%的γH2AX焦点分布在末端或中间位置看似未受损的染色质上,并且它们大小各异,因此可分为小、中、大焦点。进入有丝分裂的染色体和染色单体断裂处可发现大(60%)和中(30%)的末端γH2AX焦点,但很少由小γH2AX焦点标记。为了评估中间γH2AX焦点是否可能是错配修复的标志,在同一张玻片上进行了多色荧光原位杂交(mFISH)分析。结果表明,位于看似完整染色质上的中间γH2AX焦点并不标记错配修复位点,并且有丝分裂期间错配修复事件从未由γH2AX焦点发出信号。最后,在分析其他DNA损伤反应(DDR)因子的存在情况时,我们发现所有γH2AX焦点——无论它们是否与可见断裂重合——总是与MRE11共定位,但不与53BP1共定位。这种模式表明,这些γH2AX焦点可能是微观可见和不可见DNA损伤的标志,其中正在发生活跃但不完整或停滞的DDR。

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