Koenig Alex L, Kupper Amy E, Skidmore Jay R, Murphy Karly M
J Rehabil Res Dev. 2014;51(8):1277-86. doi: 10.1682/JRRD.2014.02.0047.
The aim of this study was to examine the relationship between biopsychosocial functioning and pain severity and to evaluate whether pain self-efficacy (PSE) mediates this relationship. This study used archival data from a multidisciplinary pain management program. Participants were 99 individuals (69% female) with chronic low back pain who completed measures of biological, psychological, and social functioning, pain severity, and PSE at admission. They ranged in age from 18 to 72 yr (mean = 42.6, standard deviation = 12.1). Structural equation modeling and bootstrapping techniques were used to test the significance of the mediated model. As we predicted, lower biological functioning (beta = -0.011; 95% confidence interval [CI] = -0.019 to -0.004, p = 0.002) and social functioning (beta = -0.009; 95% CI = -0.016 to -0.003, p = 0.007) were found to significantly predict higher pain severity, and lower social functioning was found to significantly predict lower PSE (beta = 0.196; 95% CI = -0.130 to 0.273, p = 0.002). PSE did not mediate the relationship between biopsychosocial functioning and pain severity, and psychological functioning did not significantly predict pain severity or PSE. These findings suggest that social functioning is an important factor in predicting outcomes and has a number of treatment implications.
本研究的目的是检验生物心理社会功能与疼痛严重程度之间的关系,并评估疼痛自我效能感(PSE)是否介导了这种关系。本研究使用了来自多学科疼痛管理项目的存档数据。参与者为99名患有慢性下腰痛的个体(69%为女性),他们在入院时完成了生物、心理和社会功能、疼痛严重程度以及PSE的测量。他们的年龄在18岁至72岁之间(平均 = 42.6,标准差 = 12.1)。采用结构方程模型和自抽样技术来检验中介模型的显著性。正如我们所预测的,较低的生物功能(β = -0.011;95%置信区间[CI] = -0.019至-0.004,p = 0.002)和社会功能(β = -0.009;95% CI = -0.016至-0.003,p = 0.007)被发现能显著预测更高的疼痛严重程度,并且较低的社会功能被发现能显著预测较低的PSE(β = 0.196;95% CI = -0.130至0.273,p = 0.002)。PSE并未介导生物心理社会功能与疼痛严重程度之间的关系,并且心理功能并未显著预测疼痛严重程度或PSE。这些发现表明,社会功能是预测结果的一个重要因素,并且具有许多治疗意义。