Division of Pediatric Radiology, Department of Radiology, Medical University of Graz, Auenbruggerplatz 34, 8036, Graz, Austria.
Department of Ultrasonography, First Affiliated Hospital of Xinjiang Medical University, Urumqi, Xinjiang, Peple's Republic of China, 830011.
Eur Radiol. 2015 Dec;25(12):3528-35. doi: 10.1007/s00330-015-3788-y. Epub 2015 Jul 2.
Right ventricular (RV) function represents a prognostic marker in patients with corrected congenital heart disease. In up to 80 % of these patients, right branch bundle block (RBBB) exists and leads to asynchronous ventricular contraction. The aim of this retrospective study was to evaluate the change of RV performance parameters considering delayed RV end-systolic contraction.
RV volumes of 33 patients were assessed twice: 1) not taking account of RBBB (group I), and 2) considering RBBB (group II). According to the RV ejection fraction (EF) for both groups, RV function was classified in different categories (>50 % = normal, 40-50 % = mildly-, 30-40 % = moderately-, <30 % = severely-reduced).
The mean time difference between maximal systolic contraction of the septum and RV free wall was 90.7 ms ± 42.6. Consequently, RV end-systolic volume was significantly decreased in group II (p < 0.001). Accordingly, RV stroke volume and RV EF were significantly higher in group II (p < 0.001). There was also a significant change in the assessment of RV function (p < 0.02).
RBBB induced delayed RV contraction can be detected at CMR. Ignoring the RV physiology in RBBB patients leads to a statistically significant underscoring of RV performance parameters.
• Right branch bundle block leads to an asynchronous ventricular contraction • In CMR, a delayed right ventricular contraction due to RBBB can be detected • Ignoring RV physiology in RBBB patients leads to underscoring of RV performance.
右心室(RV)功能是矫正先天性心脏病患者的预后标志物。在这些患者中,高达 80%存在右束支传导阻滞(RBBB),导致心室收缩不同步。本回顾性研究旨在评估考虑 RV 收缩末期延迟时 RV 功能参数的变化。
33 例患者的 RV 容积评估了两次:1)不考虑 RBBB(组 I),2)考虑 RBBB(组 II)。根据两组的 RV 射血分数(EF),将 RV 功能分为不同类别(>50%=正常,40-50%=轻度,30-40%=中度,<30%=严重降低)。
室间隔最大收缩与 RV 游离壁最大收缩之间的平均时间差为 90.7ms±42.6ms。因此,组 II 中 RV 收缩末期容积明显减少(p<0.001)。相应地,组 II 中 RV 每搏量和 RV EF 明显更高(p<0.001)。RV 功能评估也有显著变化(p<0.02)。
CMR 可检测到 RBBB 引起的 RV 收缩延迟。在 RBBB 患者中忽略 RV 生理学导致 RV 功能参数的统计学显著夸大。
右束支传导阻滞导致心室收缩不同步。
在 CMR 中,可以检测到 RBBB 引起的右心室收缩延迟。
在 RBBB 患者中忽略 RV 生理学导致 RV 功能参数被夸大。