Tusman Gerardo, Acosta Cecilia M, Nicola Marco, Esperatti Mariano, Bohm Stephan H, Suarez-Sipmann Fernando
Department of Anesthesia, Hospital Privado de Comunidad, Córdoba 4545, 7600, Mar del Plata, Buenos Aires, Argentina.
Intensive Care Medicine, Hospital Privado de Comunidad, Mar del Plata, Buenos Aires, Argentina.
Crit Ultrasound J. 2015 Dec;7(1):19. doi: 10.1186/s13089-015-0036-2. Epub 2015 Dec 12.
Ventilator-induced lung injury is a form of mechanical damage leading to a pulmonary inflammatory response related to the use of mechanical ventilation enhanced by the presence of atelectasis. One proposed mechanism of this injury is the repetitive opening and closing of collapsed alveoli and small airways within these atelectatic areas-a phenomenon called tidal recruitment. The presence of tidal recruitment is difficult to detect, even with high-resolution images of the lungs like CT scan. The purpose of this article is to give evidence of tidal recruitment by lung ultrasound.
A standard lung ultrasound inspection detected lung zones of atelectasis in mechanically ventilated patients. With a linear probe placed in the intercostal oblique position. We observed tidal recruitment within atelectasis as an improvement in aeration at the end of inspiration followed by the re-collapse at the end of expiration. This mechanism disappeared after the performance of a lung recruitment maneuver.
Lung ultrasound was helpful in detecting the presence of atelectasis and tidal recruitment and in confirming their resolution after a lung recruitment maneuver.
呼吸机相关性肺损伤是一种机械性损伤形式,可导致与机械通气使用相关的肺部炎症反应,肺不张会加重这种反应。这种损伤的一种推测机制是这些肺不张区域内塌陷的肺泡和小气道反复开闭——这一现象称为潮式复张。即使借助如CT扫描这样的肺部高分辨率图像,潮式复张的存在也很难检测到。本文的目的是通过肺部超声来证明潮式复张的存在。
一项标准的肺部超声检查在机械通气患者中检测到了肺不张区域。将线性探头置于肋间斜位。我们观察到肺不张内的潮式复张表现为吸气末通气改善,随后呼气末再次塌陷。在进行肺复张操作后,这一机制消失。
肺部超声有助于检测肺不张和潮式复张的存在,并确认肺复张操作后它们的消退情况。