Schwartz A N, Wang K Y, Mack L A, Lowe M, Berger R E, Cyr D R, Feldman M
Department of Radiology, University of Washington School of Medicine, Seattle 98195.
AJR Am J Roentgenol. 1989 Dec;153(6):1155-60. doi: 10.2214/ajr.153.6.1155.
To understand better the vascular events that occur during normal erection, we used color flow Doppler sonography to examine the erectile process in seven normal subjects. The relationship of systolic/diastolic velocities and spectral waveform changes in the penile arteries was studied in response to increasing pressure within the corpora cavernosa (tumescence) before and after the intracorporal administration of drugs to induce erection. A characteristic spectral waveform pattern was identified throughout the erectile cycle, and the pattern was correlated with increasing pressure. Both systolic and diastolic velocities were elevated during the initiation of erection, at which time the pressure within the corpora cavernosa was low (11-25 mm Hg). Systolic velocities remained elevated until the last phase of erection (83-106 mm Hg). Diastolic velocity decreased as the intracorporal pressure increased from 25 to 40 mm Hg. Between 40 and 63 mm Hg, diastolic velocity approximated zero. With increasing pressure (63-83 mm Hg), diastolic flow reversed. As the corpora cavernosa approached full erection (83-105 mm Hg), both the forward systolic and reverse diastolic flow components diminished. At approximately 106 mm Hg (systolic occlusion pressure), flow ceased. Systolic/diastolic velocity and waveform phase relationships could be used to define the integrity of both the cavernosal artery inflow and the venous sinusoidal outflow occlusion mechanisms. Color flow Doppler sonography enhanced our ability to observe and quantify dynamic erectile events and provided new insights into understanding normal erectile function.
为了更好地理解正常勃起过程中发生的血管事件，我们使用彩色多普勒超声检查了7名正常受试者的勃起过程。在阴茎海绵体内注射诱导勃起的药物前后，研究了阴茎动脉收缩期/舒张期速度与频谱波形变化与海绵体内压力升高（肿胀）的关系。在整个勃起周期中识别出一种特征性的频谱波形模式，并且该模式与压力升高相关。勃起开始时，海绵体内压力较低（11 - 25 mmHg），此时收缩期和舒张期速度均升高。收缩期速度在勃起的最后阶段（83 - 106 mmHg）之前一直保持升高。随着海绵体内压力从25 mmHg升高到40 mmHg，舒张期速度降低。在40至63 mmHg之间，舒张期速度接近零。随着压力升高（63 - 83 mmHg），舒张期血流逆转。当海绵体接近完全勃起（83 - 105 mmHg）时，收缩期正向血流和舒张期反向血流成分均减少。在大约106 mmHg（收缩期闭塞压力）时，血流停止。收缩期/舒张期速度和波形相位关系可用于定义海绵体动脉流入和静脉窦流出闭塞机制的完整性。彩色多普勒超声增强了我们观察和量化动态勃起事件的能力，并为理解正常勃起功能提供了新的见解。
