Kaiser Susanne M, Harms Oliver, Konar Martin, Staudacher Anne, Langer Anna, Thiel Cetina, Kramer Martin, Schaub Sebastian, von Pückler Kerstin H
Dr. med. vet. Susanne Kaiser, Department of Veterinary Clinical Science, Small Animal Clinic, Justus Liebig University, Frankfurter Straße 108, 35392 Giessen, Germany, Phone: +49 641 993 8512, Fax: +49 641 993 8509, E-mail:
Vet Comp Orthop Traumatol. 2016 Nov 23;29(6):515-521. doi: 10.3415/VCOT-16-01-0015. Epub 2016 Oct 14.
To describe clinical, radiographic, and magnetic resonance imaging (MRI) findings in 16 dogs diagnosed with gastrocnemius musculotendinopathy.
Retrospective evaluation of medical records, radiographs, and MRI results, as well as follow-up completed by telephone questionnaire.
Most dogs had chronic hindlimb lameness with no history of trauma or athletic activities. Clinical examination revealed signs of pain on palpation without stifle joint instability. Seven dogs had radiographic signs of osteophyte formation on the lateral fabella. Magnetic resonance imaging revealed T2 hyperintensity and uptake of contrast agent in the region of the origin of the gastrocnemius muscle. Changes were found in the lateral and medial heads of the gastrocnemius. Conservative treatment resulted in return to full function in 11 dogs. Two dogs showed partial restoration of normal function, one dog showed no improvement. Two dogs were lost to follow-up.
Gastrocnemius musculotendinopathy is a potential cause of chronic hindlimb lameness in medium to large breed dogs. A history of athletic activity must not necessarily be present. Magnetic resonance imaging shows signal changes and uptake of contrast agent in the region of the origin of the gastrocnemius muscle. A combination of T1 pre- and post-contrast administration and T2 weighted sequences completed by a fat-suppressed sequence in the sagittal plane are well-suited for diagnosis. Conservative treatment generally results in return to normal function.
描述16只被诊断为腓肠肌肌腱病的犬的临床、放射学及磁共振成像(MRI)表现。
对病历、X线片和MRI结果进行回顾性评估,并通过电话问卷进行随访。
大多数犬患有慢性后肢跛行,无创伤或运动活动史。临床检查发现触诊时有疼痛迹象,但膝关节无不稳定。7只犬在外侧籽骨上有骨赘形成的放射学表现。磁共振成像显示腓肠肌起点区域T2高信号及造影剂摄取。腓肠肌外侧头和内侧头均有改变。保守治疗使11只犬恢复了全部功能。2只犬部分恢复正常功能,1只犬无改善。2只犬失访。
腓肠肌肌腱病是中大型犬慢性后肢跛行的潜在原因。不一定有运动活动史。磁共振成像显示腓肠肌起点区域有信号改变及造影剂摄取。矢状面T1加权对比剂前后增强及T2加权序列联合脂肪抑制序列非常适合诊断。保守治疗一般可使功能恢复正常。