Mukherjee Soumya, Thakur Bhaskar, Tolias Christos
Department of Neurosurgery, King's College Hospital, London, UK.
BMJ Case Rep. 2016 Oct 19;2016:bcr2014208393. doi: 10.1136/bcr-2014-208393.
We report the first case of sudden-onset ipsilateral blindness following orbito-zygomatic craniotomy and clipping of a ruptured anterior communicating artery aneurysm. CT showed no new intracranial or intraorbital pathology. Visual evoked potentials testing, electroretinography and diffuse flash stimulation all indicated loss of right optic nerve function. Although the patient made an excellent neurological recovery, complete right-sided monocular blindness persisted at 6-month follow-up. We postulate that external pressure on the eyeball, resulting in posterior ischaemic optic neuropathy, was the primary cause of our patient's blindness. This has been hypothesised in the 3 previously published cases of blindness following pterional or frontal craniotomy for aneurysm repair. Intraoperatively, the surgeon must avoid unnecessary pressure on the eyeballs and handle the optic nerves with the utmost care. Incomplete understanding of the mechanisms of sudden visual loss postcraniotomy may result in under-reporting of this adverse event. Nevertheless, its seriousness warrants discussion during consent.
我们报告了首例在眶颧开颅和夹闭破裂的前交通动脉瘤后突然出现同侧失明的病例。CT显示无新的颅内或眶内病变。视觉诱发电位测试、视网膜电图和弥散闪光刺激均表明右侧视神经功能丧失。尽管患者神经功能恢复良好,但在6个月的随访中,右侧完全性单眼失明持续存在。我们推测眼球受到外部压力导致后部缺血性视神经病变是该患者失明的主要原因。在之前发表的3例因翼点或额部开颅修复动脉瘤后失明的病例中也有过这种推测。术中,外科医生必须避免对眼球施加不必要的压力,并极其小心地处理视神经。对开颅术后突然视力丧失机制的不完全理解可能导致对这一不良事件的报告不足。然而,其严重性值得在知情同意过程中进行讨论。