Jaeger B
Department of Anesthesiology, UCLA School of Medicine 90024-6909.
Cephalalgia. 1989 Sep;9(3):157-64. doi: 10.1046/j.1468-2982.1989.0903157.x.
The purpose of this investigation was to evaluate whether the pain of cervicogenic headache could be due to referred symptoms from myofascial trigger points. The presence or absence of cervical spine dysfunction was also of interest. Eleven patients with cervicogenic headaches were systematically examined for myofascial trigger points and cervical spine dysfunction. All patients had at least three myofascial trigger points on the symptomatic side. In eight of these patients, trigger point palpation clearly reproduced their headache. There were 70 myofascial trigger points (35 "very tender", 35 "tender") and 17 non-myofascial tender points on the symptomatic side, compared to 22 myofascial trigger points (one "very tender", 21 "tender") and 19 non-myofascial tender points on the asymptomatic side. These differences were statistically significant [chi-square (2df) = 22.04, p less than 0.0001]. All patients had some evidence of cervical dysfunction. Ten patients (91%) had specific segmental dysfunction of occiput on atlas and/or atlas on axis. Five patients were entered into a non-invasive, interdisciplinary pain management program designed to treat cervical spine dysfunction and myofascial pain. Treated patients reported a significant decrease in the frequency and intensity of their headaches during a median two-year follow-up. It is concluded that myofascial trigger points may be an important pain producing mechanism in cervicogenic headache and that segmental cervical dysfunction is a common feature in such patients. Conservative, non-surgical treatment appears to be effective in reducing the frequency and intensity of cervicogenic headache. These data suggest that surgical approaches should be reserved only for those patients who fail conservative therapy.
本研究的目的是评估颈源性头痛的疼痛是否可能源于肌筋膜触发点的牵涉痛症状。颈椎功能障碍的有无也受到关注。对11例颈源性头痛患者进行了肌筋膜触发点和颈椎功能障碍的系统检查。所有患者在症状侧至少有3个肌筋膜触发点。其中8例患者,触发点触诊明显重现了他们的头痛。症状侧有70个肌筋膜触发点(35个“非常敏感”,35个“敏感”)和17个非肌筋膜敏感点,无症状侧有22个肌筋膜触发点(1个“非常敏感”,21个“敏感”)和19个非肌筋膜敏感点。这些差异具有统计学意义[卡方检验(2自由度)=22.04,p<0.0001]。所有患者均有颈椎功能障碍的某些证据。10例患者(91%)存在枕骨与寰椎和/或寰椎与枢椎的特定节段功能障碍。5例患者进入了一个非侵入性的跨学科疼痛管理项目,该项目旨在治疗颈椎功能障碍和肌筋膜疼痛。在中位两年的随访期间,接受治疗的患者报告头痛的频率和强度显著降低。结论是,肌筋膜触发点可能是颈源性头痛中一个重要的疼痛产生机制,节段性颈椎功能障碍是此类患者的一个常见特征。保守的非手术治疗似乎对降低颈源性头痛的频率和强度有效。这些数据表明,手术方法应仅保留给那些保守治疗失败的患者。