Danilov N V, Aristarhov V G, Kvasov A V
Kaluga Regional Clinical Oncology Dispensary, Kaluga, 248007, Russian Federation.
Ryazan I.P.Pavlov State Medical University, Ryazan, 390026, Russian Federation;
Adv Gerontol. 2016;29(3):487-490.
In the article describes the role of deficiency of calcitonin, which originate after surgical operations on the thyroid gland, in headway of osteoporosis. Regarded significance such factors as age, level of parathyroid hormone, volume of the operational intervention in the development of osteoporosis. In elder people with thyroid nodular pathology bone metabolism is influenced by the amount of thyroid residue after resection. After thyroidectomy thyrocalcitonin is not produced, but the function of parathyroid glands remains normal, and this contributes to the progression of osteoporosis. In case of limited thyroid resection minimal changes can be observed in bone metabolism.
该文章描述了甲状腺手术后降钙素缺乏在骨质疏松症进展中的作用。还考虑了年龄、甲状旁腺激素水平、手术干预量等因素在骨质疏松症发展中的意义。在患有甲状腺结节病变的老年人中,骨代谢受切除术后甲状腺残余量的影响。甲状腺切除术后不再产生甲状腺降钙素,但甲状旁腺功能保持正常,这促使骨质疏松症进展。在甲状腺切除范围有限的情况下,骨代谢中可观察到微小变化。