Neurochemical changes in brains of mice infected with Pichinde virus.

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作者信息

Barrett A D, Johnson J A, Cross A J, Taylor G R

出版信息

Med Microbiol Immunol. 1986;175(2-3):213-5. doi: 10.1007/BF02122454.

DOI:10.1007/BF02122454

PMID:2873503

Abstract

摘要

相似文献

Neurochemical changes in brains of mice infected with Pichinde virus.

Med Microbiol Immunol. 1986;175(2-3):213-5. doi: 10.1007/BF02122454.

Subclinical infections in mice resulting from the modulation of a lethal dose of Semliki Forest virus with defective interfering viruses: neurochemical abnormalities in the central nervous system.用缺陷干扰病毒调节致死剂量的塞姆利基森林病毒后小鼠发生的亚临床感染：中枢神经系统的神经化学异常

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Persistence of virulent Semliki Forest virus in mouse brain following co-inoculation with defective interfering particles.与缺陷干扰颗粒共同接种后，强毒株塞姆利基森林病毒在小鼠脑中的持续存在。

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Production of defective interfering virus in the brains of mice by an avirulent, in contrast with a virulent, strain of Semliki forest virus.与毒力强的塞姆利基森林病毒株相比，无毒力的塞姆利基森林病毒株在小鼠大脑中产生缺陷干扰病毒。

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Pathogenesis of Pichinde virus infection in strain 13 guinea pigs: an immunocytochemical, virologic, and clinical chemistry study.13 品系豚鼠感染皮钦德病毒的发病机制：一项免疫细胞化学、病毒学及临床化学研究。

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本文引用的文献

The concurrent estimation of the major monoamine metabolites in human and non-human primate brain by HPLC with fluorescence and electrochemical detection.

Life Sci. 1981 Feb 2;28(5):499-505. doi: 10.1016/0024-3205(81)90143-0.

Radiochemical micro assays for the determination of choline acetyltransferase and acetylcholinesterase activities.用于测定胆碱乙酰转移酶和乙酰胆碱酯酶活性的放射化学微量分析。

Biochem J. 1969 Nov;115(3):465-72. doi: 10.1042/bj1150465.

The monoamine metabolism in viral encephalitides of the mouse. I. Virological and biochemical results.小鼠病毒性脑炎中的单胺代谢。I. 病毒学和生化结果。

Brain Res. 1970 Oct 13;23(2):235-46. doi: 10.1016/0006-8993(70)90042-9.

Alterations of acetylcholine enzymes in neuroblastoma cells persistently infected with lymphocytic choriomeningitis virus.持续感染淋巴细胞性脉络丛脑膜炎病毒的神经母细胞瘤细胞中乙酰胆碱酶的变化。

J Cell Physiol. 1977 Jun;91(3):459-72. doi: 10.1002/jcp.1040910316.

Neurochemical changes following kainic acid lesions of the nucleus accumbens: implications for a GABAergic accumbal-ventral tegmental pathway.海人酸损伤伏隔核后的神经化学变化：对γ-氨基丁酸能伏隔核-腹侧被盖区通路的影响

Life Sci. 1978 Mar;22(11):1011-4. doi: 10.1016/0024-3205(78)90367-3.

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