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Glutamate dysregulation and glutamatergic therapeutics for PTSD: Evidence from human studies.
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Methylone promotes neurite outgrowth and has long-lasting effects on fear extinction learning.
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Post-Stroke PTSD: The Protective Role of CCR5-Δ32 Polymorphism.
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Clinical conceptualisation of PTSD in psilocybin treatment: disrupting a pre-determined and over-determined maladaptive interpretive framework.
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Non-response to short-term ketamine use for treatment-resistant depression.
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Advances in synaptic PET imaging and intervention with synapse-targeted small-molecular drugs for dementia diagnosis and therapy.
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Microglia-mediated neuroimmune suppression in PTSD is associated with anhedonia.
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Challenges and rewards of in vivo synaptic density imaging, and its application to the study of depression.
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Findings of PTSD-specific deficits in default mode network strength following a mild experimental stressor.
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本文引用的文献

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Morphovolumetric changes after EMDR treatment in drug-naïve PTSD patients.
Riv Psichiatr. 2017 Jan-Feb;52(1):24-31. doi: 10.1708/2631.27051.
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Cortical thickness reduction in combat exposed U.S. veterans with and without PTSD.
Eur Neuropsychopharmacol. 2017 May;27(5):515-525. doi: 10.1016/j.euroneuro.2017.02.010. Epub 2017 Mar 6.
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Pretreatment biomarkers predicting PTSD psychotherapy outcomes: A systematic review.
Neurosci Biobehav Rev. 2017 Apr;75:140-156. doi: 10.1016/j.neubiorev.2017.01.027. Epub 2017 Jan 28.
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Ketamine accelerates fear extinction via mTORC1 signaling.
Neurobiol Dis. 2017 Apr;100:1-8. doi: 10.1016/j.nbd.2016.12.026. Epub 2016 Dec 30.
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Glutamate dysregulation and glutamatergic therapeutics for PTSD: Evidence from human studies.
Neurosci Lett. 2017 May 10;649:147-155. doi: 10.1016/j.neulet.2016.11.064. Epub 2016 Dec 1.
7
Chronic stress and hippocampal dendritic complexity: Methodological and functional considerations.
Physiol Behav. 2017 Sep 1;178:66-81. doi: 10.1016/j.physbeh.2016.11.017. Epub 2016 Nov 22.
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Brain connectivity and neurological disorders after stroke.
Curr Opin Neurol. 2016 Dec;29(6):706-713. doi: 10.1097/WCO.0000000000000396.
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Context Processing and the Neurobiology of Post-Traumatic Stress Disorder.
Neuron. 2016 Oct 5;92(1):14-30. doi: 10.1016/j.neuron.2016.09.039.
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Inflammation, Glutamate, and Glia: A Trio of Trouble in Mood Disorders.
Neuropsychopharmacology. 2017 Jan;42(1):193-215. doi: 10.1038/npp.2016.199. Epub 2016 Sep 15.

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