Cardiac pericyte is promising target for ischemic heart diseases: Role of Notch3.
作者信息
Chen Jian-Xiong, Chen Sean T, Tao Yong-Kang
机构信息
Department of Pharmacology and Toxicology, University of Mississippi Medical Center, School of Medicine, Jackson, MS 39216, USA.
Duke University School of Medicine, Durham, USA.
出版信息
Int J Cardiol. 2017 Nov 1;246:57. doi: 10.1016/j.ijcard.2017.05.056.
Abstract
摘要
相似文献
1
Cardiac pericyte is promising target for ischemic heart diseases: Role of Notch3.
Int J Cardiol. 2017 Nov 1;246:57. doi: 10.1016/j.ijcard.2017.05.056.
2
Discovering cardiac pericyte biology: From physiopathological mechanisms to potential therapeutic applications in ischemic heart disease.
Vascul Pharmacol. 2016 Nov;86:53-63. doi: 10.1016/j.vph.2016.05.009. Epub 2016 Jun 5.
3
The fate and role of the pericytes in myocardial diseases.
Eur J Clin Invest. 2024 Aug;54(8):e14204. doi: 10.1111/eci.14204. Epub 2024 Apr 8.
4
A potential and novel therapeutic approach to ischemic heart diseases: Notch3.
Int J Cardiol. 2017 Nov 1;246:58. doi: 10.1016/j.ijcard.2017.04.093.
5
The role of cardiac pericytes in health and disease: therapeutic targets for myocardial infarction.
Nat Rev Cardiol. 2024 Feb;21(2):106-118. doi: 10.1038/s41569-023-00913-y. Epub 2023 Aug 4.
6
Pericytes in the infarcted heart.
Vasc Biol. 2019 Apr 25;1(1):H23-H31. doi: 10.1530/VB-19-0007. eCollection 2019.
7
LPS causes pericyte loss and microvascular dysfunction via disruption of Sirt3/angiopoietins/Tie-2 and HIF-2α/Notch3 pathways.
Sci Rep. 2016 Feb 12;6:20931. doi: 10.1038/srep20931.
8
Cardiac Pericyte Diversity in Infarct Remodeling: Not Just Vascular Support Cells?
Circulation. 2023 Sep 12;148(11):899-902. doi: 10.1161/CIRCULATIONAHA.123.065676. Epub 2023 Sep 11.
9
Mechanisms of fibrosis: the role of the pericyte.
Curr Opin Nephrol Hypertens. 2011 May;20(3):297-305. doi: 10.1097/MNH.0b013e328344c3d4.
10
Notch3 Ameliorates Cardiac Fibrosis After Myocardial Infarction by Inhibiting the TGF-β1/Smad3 Pathway.
Cardiovasc Toxicol. 2016 Oct;16(4):316-24. doi: 10.1007/s12012-015-9341-z.
引用本文的文献
1
Nestin and Notch3 collaboratively regulate angiogenesis, collagen production, and endothelial-mesenchymal transition in lung endothelial cells.
Cell Commun Signal. 2023 Sep 21;21(1):247. doi: 10.1186/s12964-023-01099-z.
2
Matters of the heart: Cellular sex differences.
J Mol Cell Cardiol. 2021 Nov;160:42-55. doi: 10.1016/j.yjmcc.2021.04.010. Epub 2021 Jun 22.
3
Emerging Role of Pericytes and Their Secretome in the Heart.
Cells. 2021 Mar 4;10(3):548. doi: 10.3390/cells10030548.
4
Sirtuin 3, Endothelial Metabolic Reprogramming, and Heart Failure With Preserved Ejection Fraction.
J Cardiovasc Pharmacol. 2019 Oct;74(4):315-323. doi: 10.1097/FJC.0000000000000719.
本文引用的文献
1
LPS causes pericyte loss and microvascular dysfunction via disruption of Sirt3/angiopoietins/Tie-2 and HIF-2α/Notch3 pathways.
Sci Rep. 2016 Feb 12;6:20931. doi: 10.1038/srep20931.
2
Notch3 Ameliorates Cardiac Fibrosis After Myocardial Infarction by Inhibiting the TGF-β1/Smad3 Pathway.
Cardiovasc Toxicol. 2016 Oct;16(4):316-24. doi: 10.1007/s12012-015-9341-z.
3
Notch3/Akt signaling contributes to OSM-induced protection against cardiac ischemia/reperfusion injury.
Apoptosis. 2015 Sep;20(9):1150-63. doi: 10.1007/s10495-015-1148-7.
4
A role for pericytes in coronary no-reflow.
Nat Rev Cardiol. 2014 Jul;11(7):427-32. doi: 10.1038/nrcardio.2014.58. Epub 2014 Apr 29.
5
Targeting pericyte differentiation as a strategy to modulate kidney fibrosis in diabetic nephropathy.
Semin Nephrol. 2012 Sep;32(5):463-70. doi: 10.1016/j.semnephrol.2012.07.009.
Zotero 插件