Grobet P, Gilon Y, Bruwier A, Nizet J-L
Service de Chirurgie Plastique et Maxillo-faciale, CHU de Liège, Site Sart Tilman, Liège, Belgique.
Service d'Orthodontie et Orthopédie Dento-faciale, CHU de Liège, Site Sart Tilman, Liège, Belgique.
Rev Med Liege. 2017 Sep;72(9):410-415.
Although well studied since the 50's, bruxism remains a largely unknown pathology. Its origin is complex, mixing psychological as well as neurological, odontological and hypnic aspects. However, the few analyzes performed on this topic have allowed to set convincing etiopathological hypotheses, including central dysregulation of the dopaminergic system as well as of the neuro-masticatory system. To avoid harmful consequences as headaches, temporomandibular disorders and premature dental scuffs / fractures, it is mandatory to diagnose bruxism as early as possible. For this purpose, and in addition to anamnestic and clinical data, the practitioner can confirm diagnosis with polysomnography, including electromyographic study of masticatory muscles and audiovisual recording. Some orthodontic, pharmacological and psychological solutions have already proved efficient. Nevertheless, a better knowledge of causative neurobiological mechanisms would allow to foresee etiology-based treatments.
尽管自50年代以来就对磨牙症进行了深入研究,但它在很大程度上仍是一种鲜为人知的病症。其病因复杂,涉及心理、神经、牙科学和睡眠等多个方面。然而,针对这一主题进行的少量分析已得出了令人信服的病因病理假设,包括多巴胺能系统以及神经咀嚼系统的中枢调节异常。为避免出现头痛、颞下颌关节紊乱以及牙齿过早磨损/折断等有害后果,必须尽早诊断磨牙症。为此,除了问诊和临床数据外,医生还可通过多导睡眠图来确诊,包括咀嚼肌的肌电图研究和视听记录。一些正畸、药物和心理治疗方法已被证明有效。然而,更深入地了解致病的神经生物学机制将有助于预见基于病因的治疗方法。