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Mice deficient for ERAD machinery component Sel1L develop central diabetes insipidus.
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ER-associated degradation is required for vasopressin prohormone processing and systemic water homeostasis.
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Association of the SEL1L protein transmembrane domain with HRD1 ubiquitin ligase regulates ERAD-L.
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PDI reductase acts on Akita mutant proinsulin to initiate retrotranslocation along the Hrd1/Sel1L-p97 axis.
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SEL1L-dependent Substrates Require Derlin2/3 and Herp1/2 for Endoplasmic Reticulum-associated Degradation.
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Role of HERP and a HERP-related protein in HRD1-dependent protein degradation at the endoplasmic reticulum.
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Hepatic Sel1L-Hrd1 ER-associated degradation (ERAD) manages FGF21 levels and systemic metabolism via CREBH.
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本文引用的文献

1
ER-associated degradation is required for vasopressin prohormone processing and systemic water homeostasis.
J Clin Invest. 2017 Oct 2;127(10):3897-3912. doi: 10.1172/JCI94771. Epub 2017 Sep 18.
2
Neural circuits underlying thirst and fluid homeostasis.
Nat Rev Neurosci. 2017 Aug;18(8):459-469. doi: 10.1038/nrn.2017.71. Epub 2017 Jun 22.
3
New Insights into the Physiological Role of Endoplasmic Reticulum-Associated Degradation.
Trends Cell Biol. 2017 Jun;27(6):430-440. doi: 10.1016/j.tcb.2016.12.002. Epub 2017 Feb 9.
5
Genetic forms of neurohypophyseal diabetes insipidus.
Best Pract Res Clin Endocrinol Metab. 2016 Mar;30(2):249-62. doi: 10.1016/j.beem.2016.02.008. Epub 2016 Feb 18.
6
INS-gene mutations: from genetics and beta cell biology to clinical disease.
Mol Aspects Med. 2015 Apr;42:3-18. doi: 10.1016/j.mam.2014.12.001. Epub 2014 Dec 24.
7
Sel1L is indispensable for mammalian endoplasmic reticulum-associated degradation, endoplasmic reticulum homeostasis, and survival.
Proc Natl Acad Sci U S A. 2014 Feb 4;111(5):E582-91. doi: 10.1073/pnas.1318114111. Epub 2014 Jan 22.
8
The mammalian endoplasmic reticulum-associated degradation system.
Cold Spring Harb Perspect Biol. 2013 Sep 1;5(9):a013185. doi: 10.1101/cshperspect.a013185.

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