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针对癌症中的 PI3K 通路:我们是否取得进展?

Targeting the PI3K pathway in cancer: are we making headway?

机构信息

Department of Investigational Cancer Therapeutics (Phase I Clinical Trials Program), The University of Texas MD Anderson Cancer Center, Houston, TX, USA.

出版信息

Nat Rev Clin Oncol. 2018 May;15(5):273-291. doi: 10.1038/nrclinonc.2018.28. Epub 2018 Mar 6.


DOI:10.1038/nrclinonc.2018.28
PMID:29508857
Abstract

The PI3K-AKT-mTOR pathway is one of the most frequently dysregulated pathways in cancer and, consequently, more than 40 compounds that target key components of this signalling network have been tested in clinical trials involving patients with a range of different cancers. The clinical development of many of these agents, however, has not advanced to late-phase randomized trials, and the antitumour activity of those that have been evaluated in comparative prospective studies has typically been limited, or toxicities were found to be prohibitive. Nevertheless, the mTOR inhibitors temsirolimus and everolimus and the PI3K inhibitors idelalisib and copanlisib have been approved by the FDA for clinical use in the treatment of a number of different cancers. Novel compounds with greater potency and selectivity, as well as improved therapeutic indices owing to reduced risks of toxicity, are clearly required. In addition, biomarkers that are predictive of a response, such as PIK3CA mutations for inhibitors of the PI3K catalytic subunit α isoform, must be identified and analytically and clinically validated. Finally, considering that oncogenic activation of the PI3K-AKT-mTOR pathway often occurs alongside pro-tumorigenic aberrations in other signalling networks, rational combinations are also needed to optimize the effectiveness of treatment. Herein, we review the current experience with anticancer therapies that target the PI3K-AKT-mTOR pathway.

摘要

PI3K-AKT-mTOR 通路是癌症中最常失调的通路之一,因此,已有超过 40 种针对该信号网络关键组成部分的化合物在涉及多种不同癌症患者的临床试验中进行了测试。然而,这些药物中的许多在临床开发中并未进展到后期随机试验,并且在比较性前瞻性研究中评估的那些药物的抗肿瘤活性通常受到限制,或者发现毒性是不可接受的。尽管如此,mTOR 抑制剂替西罗莫司和依维莫司以及 PI3K 抑制剂idelalisib 和 copanlisib 已被 FDA 批准用于治疗多种不同的癌症。显然,需要具有更高效力和选择性的新型化合物,以及由于降低毒性风险而导致治疗指数改善的化合物。此外,需要鉴定预测反应的生物标志物,例如针对 PI3K 催化亚基 α 同工型抑制剂的 PIK3CA 突变,并对其进行分析和临床验证。最后,考虑到致癌激活 PI3K-AKT-mTOR 通路通常与其他信号网络中的促肿瘤发生异常同时发生,也需要合理的组合来优化治疗效果。本文综述了针对 PI3K-AKT-mTOR 通路的抗癌治疗的当前经验。

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Targeting the PI3K pathway in cancer: are we making headway?

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本文引用的文献

[1]
Phosphatidylinositol 3-Kinase α-Selective Inhibition With Alpelisib (BYL719) in PIK3CA-Altered Solid Tumors: Results From the First-in-Human Study.

J Clin Oncol. 2018-2-5

[2]
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Lancet Oncol. 2017-12-7

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A Phase I Open-Label Study to Identify a Dosing Regimen of the Pan-AKT Inhibitor AZD5363 for Evaluation in Solid Tumors and in -Mutated Breast and Gynecologic Cancers.

Clin Cancer Res. 2017-10-24

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Clin Cancer Res. 2017-10-19

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Cancer Treat Rev. 2017-7-18

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Clin Cancer Res. 2017-6-23

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Lancet Oncol. 2017-7

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