Walley Kempland C, Roush Evan P, Stauch Christopher M, Kunselman Allen R, Saloky Kaitlin L, King Jesse L, Lewis Gregory S, Aynardi Michael C
Department of Orthopaedic Surgery, Penn State Hershey Bone and Joint Institute (KCW, EPR, CMS, KLS, JLK, GSL, MCA) Penn State Milton S. Hershey Medical Center, Hershey, Pennsylvania.
Department of Public Health Sciences (ARK) Penn State Milton S. Hershey Medical Center, Hershey, Pennsylvania.
Foot Ankle Spec. 2019 Aug;12(4):316-321. doi: 10.1177/1938640018796618. Epub 2018 Aug 31.
The pathophysiology of adult-acquired flatfoot deformity (AAFD) is not fully explained by degeneration of the posterior tibial tendon alone. While a shortened or dysplastic lateral column has been implicated in flatfoot deformity in pediatrics, there is no study that has quantified the degree of dysplasia in adults with a stage IIb flatfoot deformity, or if any exists at all. : An institutional radiology database was queried for patients with posterior tibial tendon dysfunction (PTTD) who had computed tomography (CT) performed. Controls were patients receiving CT scan for an intra-articular distal tibia fracture without preexisting foot or calcaneal pathology. Clinical notes, physical examination, and weightbearing radiographs were used to find patients that met clinical criteria for stage IIb PTTD. Morphometric measurements of the calcanei were performed involving the length of the calcaneal axis (LCA), height of the anterior process (HAP), and length of the anterior process (LAP). All measurements were performed independently by separate observers, with observers blinded to group assignment. We considered a difference of ±4 mm as our threshold. 7 patients and 7 controls were available for reconstruction and analysis. On average, the LCA was 3.1 mm shorter in patients with stage IIb PTTD compared with controls ( < .05). The LAP was shorter in PTTD patients compared with controls 3.4 mm ( < .001). Our results support the hypothesis that the calcaneus of adult patients with stage IIb AAFD is dysplastic when compared with healthy controls, which further supports the utility of lateral column lengthening. Level III: Case-control study.
成人获得性平足畸形(AAFD)的病理生理学不能仅通过胫后肌腱退变来完全解释。虽然外侧柱缩短或发育异常与小儿扁平足畸形有关,但尚无研究对IIb期扁平足畸形成人的发育异常程度进行量化,或者是否存在发育异常。查询机构放射学数据库,寻找接受过计算机断层扫描(CT)的胫后肌腱功能障碍(PTTD)患者。对照组为接受CT扫描的胫骨干远端关节内骨折患者,且无足部或跟骨既往病变。通过临床记录、体格检查和负重X线片来找出符合IIb期PTTD临床标准的患者。对跟骨进行形态测量,包括跟骨轴长度(LCA)、前突高度(HAP)和前突长度(LAP)。所有测量均由不同的观察者独立进行,观察者对分组情况不知情。我们将±4mm的差异作为阈值。7例患者和7例对照可供重建和分析。平均而言,IIb期PTTD患者的LCA比对照组短3.1mm(P<0.05)。PTTD患者的LAP比对照组短3.4mm(P<0.001)。我们的结果支持以下假设:与健康对照组相比,IIb期AAFD成年患者的跟骨发育异常,这进一步支持了外侧柱延长术的实用性。III级:病例对照研究。