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本文引用的文献
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Discoidin Domain Receptor 1 (DDR1) Is Necessary for Tissue Homeostasis in Pancreatic Injury and Pathogenesis of Pancreatic Ductal Adenocarcinoma.
Am J Pathol. 2020 Aug;190(8):1735-1751. doi: 10.1016/j.ajpath.2020.03.020. Epub 2020 Apr 24.
2
2-Amino-2,3-dihydro-1-indene-5-carboxamide-Based Discoidin Domain Receptor 1 (DDR1) Inhibitors: Design, Synthesis, and in Vivo Antipancreatic Cancer Efficacy.
J Med Chem. 2019 Aug 22;62(16):7431-7444. doi: 10.1021/acs.jmedchem.9b00365. Epub 2019 Aug 2.
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Getting a grip on adhesion: Cadherin switching and collagen signaling.
Biochim Biophys Acta Mol Cell Res. 2019 Nov;1866(11):118472. doi: 10.1016/j.bbamcr.2019.04.002. Epub 2019 Apr 5.
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Stromal Content Is Correlated With Tissue Site, Contrast Retention, and Survival in Pancreatic Adenocarcinoma.
JCO Precis Oncol. 2018;2018. doi: 10.1200/PO.17.00121. Epub 2018 Jan 16.
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Stromal biology and therapy in pancreatic cancer: ready for clinical translation?
Gut. 2019 Jan;68(1):159-171. doi: 10.1136/gutjnl-2018-316451. Epub 2018 Sep 3.
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A Visually Apparent and Quantifiable CT Imaging Feature Identifies Biophysical Subtypes of Pancreatic Ductal Adenocarcinoma.
Clin Cancer Res. 2018 Dec 1;24(23):5883-5894. doi: 10.1158/1078-0432.CCR-17-3668. Epub 2018 Aug 6.
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Therapeutic developments in pancreatic cancer: current and future perspectives.
Nat Rev Gastroenterol Hepatol. 2018 Jun;15(6):333-348. doi: 10.1038/s41575-018-0005-x.
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Discoidin domain receptor 1 (DDR1) ablation promotes tissue fibrosis and hypoxia to induce aggressive basal-like breast cancers.
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Malnutrition in Pancreatic Ductal Adenocarcinoma (PDA): Dietary Pancreatic Enzymes Improve Short-Term Health but Stimulate Tumor Growth.
Am J Pathol. 2018 Mar;188(3):616-626. doi: 10.1016/j.ajpath.2017.11.014. Epub 2017 Dec 15.
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Identification of cancer genes that are independent of dominant proliferation and lineage programs.
Proc Natl Acad Sci U S A. 2017 Dec 26;114(52):E11276-E11284. doi: 10.1073/pnas.1714877115. Epub 2017 Dec 11.
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