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一氧化碳中毒后脑损伤延迟

Delayed brain injury post carbon monoxide poisoning.

作者信息

Mazo Jahinover, Mukhtar Eyas, Mazo Yuliya, Nagaraj Abhiram, Mantello Michael T

机构信息

Richmond University Medical Center, Department of Radiology, 355 Bard Ave Staten Island, NY 10310 USA.

出版信息

Radiol Case Rep. 2020 Aug 10;15(10):1845-1848. doi: 10.1016/j.radcr.2020.07.048. eCollection 2020 Oct.

Abstract

A 66-year-old male was found unresponsive and diagnosed with acute carbon monoxide poisoning, with pathognomonic findings on radiological imaging. During his first day's the patient underwent acute neurological deterioration; however, this was followed 2 weeks later with a subsequent improvement to near baseline. The improvement back to baseline was short-lived, and the patient quickly worsened and underwent neurological decompensation. These findings were consistent with delayed post hypoxic leukoencephalopathy, serious sequelae of carbon monoxide poisoning. This case report shows the importance of recognition of carbon monoxide toxicity and aims to improve accurate diagnosis of the sequelae that may follow using computed tomography and magnetic resonance imaging sequences, magnetic resonance spectroscopy in order to prevent or ameliorate further neurological decline.

摘要

一名66岁男性被发现无反应,诊断为急性一氧化碳中毒,影像学检查有特征性表现。在病程第一天,患者出现急性神经功能恶化;然而,2周后病情有所改善,接近基线水平。恢复到基线水平的改善是短暂的,患者很快病情恶化并出现神经失代偿。这些发现与迟发性缺氧性白质脑病相符,这是一氧化碳中毒的严重后遗症。本病例报告显示了认识一氧化碳毒性的重要性,旨在提高对可能出现的后遗症的准确诊断,可通过计算机断层扫描、磁共振成像序列、磁共振波谱分析来预防或改善进一步的神经功能衰退。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c78d/7424168/90c4c3370327/gr1.jpg

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