Cambridge Institute for Medical Research (CIMR), University of Cambridge, Cambridge, UK.
Division of Respiratory Medicine, Dept of Medicine, University of Cambridge, Cambridge, UK.
Eur Respir Rev. 2020 Oct 1;29(157). doi: 10.1183/16000617.0184-2020. Print 2020 Sep 30.
The respiratory tract and its resident immune cells face daily exposure to stress, both from without and from within. Inhaled pathogens, including severe acute respiratory syndrome coronavirus 2, and toxins from pollution trigger a cellular defence system that reduces protein synthesis to minimise viral replication or the accumulation of misfolded proteins. Simultaneously, a gene expression programme enhances antioxidant and protein folding machineries in the lung. Four kinases (PERK, PKR, GCN2 and HRI) sense a diverse range of stresses to trigger this "integrated stress response". Here we review recent advances identifying the integrated stress response as a critical pathway in the pathogenesis of pulmonary diseases, including pneumonias, thoracic malignancy, pulmonary fibrosis and pulmonary hypertension. Understanding the integrated stress response provides novel targets for the development of therapies.
呼吸道及其常驻免疫细胞每天都面临着来自内外两方面的压力。吸入的病原体,包括严重急性呼吸综合征冠状病毒 2 和来自污染的毒素,会触发细胞防御系统,减少蛋白质合成,以最大程度地减少病毒复制或错误折叠蛋白质的积累。同时,基因表达程序会增强肺部的抗氧化剂和蛋白质折叠机制。四种激酶(PERK、PKR、GCN2 和 HRI)可以感知多种应激,从而触发这种“综合应激反应”。在这里,我们回顾了最近的进展,这些进展确定了综合应激反应是肺炎、胸部恶性肿瘤、肺纤维化和肺动脉高压等肺部疾病发病机制中的关键途径。了解综合应激反应为治疗方法的开发提供了新的靶点。
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