Tin dioxide decorated on Ni-encapsulated nitrogen-doped carbon nanotubes for anodic electrolysis and persulfate activation to degrade cephalexin: Mineralization and degradation pathway.

In this study, bamboo-shaped carbon nanotubes exhibiting high nitrogen content and Ni encapsulation (Ni@NCNT) were effectively synthesized by a simple pyrolysis method. The catalytic peroxydisulfate activation for cephalexin (CPX) degradation was investigated using the prepared material. SnO was further decorated and fabricated on the anode material (SnO/Ni@NCNT) for electrochemical degradation of CPX in an aqueous solution. Transmission electron microscopy, X-ray diffraction, and X-ray photoelectron spectroscopy indicated that the SnO nanoparticles were uniformly distributed on the surface of Ni@NCNT. Electrochemical characterization employing cyclic voltammetry and linear sweep voltammetry demonstrated that SnO/Ni@NCNT displayed higher oxygen evolution potential and electrocatalytic activity than Ni@NCNT. Mineralization of CPX in wastewater was performed using electrolysis coupled with persulfate oxidation. The analysis revealed a synergistic strengthening effect. The electropersulfate oxidation resulted in higher total organic carbon (TOC) removal (70.3%) than the sum of electrooxidation (48.1%) and persulfate oxidation (9.2%) toward CPX. This phenomenon might result from the regeneration of sulfate radicals (SO) on the anode and complementary oxidation by SO and OH. Persulfate oxidation alone was shown to result in low TOC removal, although CPX was mostly degraded. Additionally, the CPX degradation pathway involving electropersulfate oxidation was proposed and it is indicated that CPX molecules were completed decomposed by the examination of short chain acids, mineralized ions, and ecotoxicity evolution indicated that the antibiotic was completely degraded. This study provides a new approach for the design and preparation of novel electrode materials and electrochemical degradation facilities for the removal of pollutants via persulfate activation.